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Cervical spinal stenosis is one of the most common forms of spinal stenosis, along with lumbar spinal stenosis (which occurs at the level of the lower back instead of the neck). Thoracic spinal stenosis, at the level of the mid-back, is much less common. [2] Cervical spinal stenosis can be far more dangerous by compressing the spinal cord.
When the plaque does not cause symptoms, people are still at higher risk of stroke than the general population, but not as high as people with symptomatic stenosis. The incidence of stroke, including fatal stroke, is 1–2% per year. The surgical mortality of endarterectomy ranges from 1–2% to as much as 10%.
Lumbar stenosis is the most common, followed by cervical stenosis. [2] Diagnosis is generally based on symptoms and medical imaging. [4] Treatment may involve medications, bracing, or surgery. [6] Medications may include NSAIDs, acetaminophen, anticonvulsants (gabapentinoids) or steroid injections.
The risk-reduction from intervention for carotid stenosis (stenting or endarterectomy) is greatest when the indication for intervention is symptoms (i.e., the patient is symptomatic) - typically stroke or TIA. [6] A new generation of double-layer stents is currently being developed to reduce the risk of stroke during or after the procedure.
Carotid artery dissection can occur spontaneously or be triggered by trauma, including minor injuries, certain medical conditions, or activities that involve neck movement. It is a leading cause of stroke in young and middle-aged adults.
In the lumbar spine it is commonly used to treat spinal claudication caused by spinal stenosis, and is considered the most effective treatment for this condition based on current evidence. [3] In the cervical and thoracic spine it is used to treat myelopathy caused by compression of the spinal cord itself.
A 37-year-old man woke up with neck pain, nausea, blurry vision. He was having a stroke caused by a cavernous malformation. After brain surgery, he is back hiking.
[48] [51] [52] MSC treatment also appears to improve the control of cerebral blood flow and blood–brain barrier permeability, [53] [54] as well as what is currently thought to be the most important mechanism of MSC treatment after stroke, the activation of endogenous neuroprotection and neurorestoration pathways by the release of cytokines ...