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  2. Saturation mutagenesis - Wikipedia

    en.wikipedia.org/wiki/Saturation_mutagenesis

    Additionally, it is usual to use degenerate codons that minimise stop codons (which are generally not desired). Consequently, the fully randomised 'NNN' is not ideal, and alternative, more restricted degenerate codons are used. 'NNK' and 'NNS' have the benefit of encoding all 20 amino acids, but still encode a stop codon 3% of the time.

  3. Codon degeneracy - Wikipedia

    en.wikipedia.org/wiki/Codon_degeneracy

    A nucleotide substitution at a 4-fold degenerate site is always a synonymous mutation with no change on the amino acid. [2]: 521–522 A less degenerate site would produce a nonsynonymous mutation on some of the substitutions. An example (and the only) 3-fold degenerate site is the third position of an isoleucine codon.

  4. Codon usage bias - Wikipedia

    en.wikipedia.org/wiki/Codon_usage_bias

    Codon usage bias in Physcomitrella patens. Codon usage bias refers to differences in the frequency of occurrence of synonymous codons in coding DNA.A codon is a series of three nucleotides (a triplet) that encodes a specific amino acid residue in a polypeptide chain or for the termination of translation (stop codons).

  5. Degeneracy (biology) - Wikipedia

    en.wikipedia.org/wiki/Degeneracy_(biology)

    Examples of degeneracy are found in the genetic code, when many different nucleotide sequences encode the same polypeptide; in protein folding, when different polypeptides fold to be structurally and functionally equivalent; in protein functions, when overlapping binding functions and similar catalytic specificities are observed; in metabolism, when multiple, parallel biosynthetic and ...

  6. Missense mutation - Wikipedia

    en.wikipedia.org/wiki/Missense_mutation

    In the most common variant of sickle-cell disease, the 20th nucleotide of the gene for the beta chain of hemoglobin is altered from the codon GAG to GTG. Thus, the 6th amino acid glutamic acid is substituted by valine —notated as an "E6V" mutation—and the protein is sufficiently altered to cause the sickle-cell disease.

  7. Crick, Brenner et al. experiment - Wikipedia

    en.wikipedia.org/wiki/Crick,_Brenner_et_al...

    The Crick, Brenner et al. experiment (1961) was a scientific experiment performed by Francis Crick, Sydney Brenner, Leslie Barnett and R.J. Watts-Tobin. It was a key experiment in the development of what is now known as molecular biology and led to a publication entitled "The General Nature of the Genetic Code for Proteins" and according to the historian of Science Horace Judson is "regarded ...

  8. Ambush hypothesis - Wikipedia

    en.wikipedia.org/wiki/Ambush_hypothesis

    The ambush hypothesis is a hypothesis in the field of molecular genetics that suggests that the prevalence of “hidden” or off-frame stop codons in DNA selectively deters off-frame translation of mRNA to save energy, molecular resources, and to reduce strain on biosynthetic machinery by truncating the production of non-functional, potentially cytotoxic protein products. [1]

  9. Silent mutation - Wikipedia

    en.wikipedia.org/wiki/Silent_mutation

    The premature insertion of a stop codon, a nonsense mutation, can alter the primary structure of a protein. [12] In this case, a truncated protein is produced. Protein function and folding is dependent on the position in which the stop codon was inserted and the amount and composition of the sequence lost.