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Nystagmus as seen in a case of ocular albinism. The cause of pathological nystagmus may be congenital, idiopathic, or secondary to a pre-existing neurological disorder.It also may be induced temporarily by disorientation (such as on roller coaster rides or when a person has been spinning in circles) or by some drugs (alcohol, lidocaine, and other central nervous system depressants, inhalant ...
This condition is characterized as a synkinesis: when two or more muscles that are independently innervated have either simultaneous or coordinated movements. [1] [2] Common physiologic examples of synkineses occur during sucking, chewing, or conjugate eye movements. There are also several abnormal cranial nerve synkineses, both acquired and ...
Fast nystagmus is the second constituent eye movement in OKR. It consists of a rapid, resetting saccade in the opposite direction of the slow nystagmus (i.e., opposite to the stimulus motion). The purpose of the fast nystagmus is to keep the eye centered in the orbit, while the purpose of the slow nystagmus is to stabilize the moving visual ...
The symptoms of vestibulocerebellar syndrome vary among patients but are typically a unique combination of ocular abnormalities including nystagmus, poor or absent smooth pursuit (ability of the eyes to follow a moving object), strabismus (misalignment of the eyes), diplopia (double vision), oscillopsia (the sensation that stationary objects in the visual field are oscillating) and abnormal ...
Parinaud's syndrome is a cluster of abnormalities of eye movement and pupil dysfunction, characterized by: Paralysis of upwards gaze: Downward gaze is usually preserved. This vertical palsy is supranuclear, so doll's head maneuver should elevate the eyes, but eventually all upward gaze mechanisms fail. In the extreme form, conjugate down gaze ...
Two images of the same child. The top image shows the left eye slightly cross-eyed. This is fixed in the bottom picture by stretching the extra skin around the eye. A deviated gaze is an abnormal movement of the eyes. It is often found as a symptom for subdural hematoma or some people may have it from birth.
A horizontal gaze palsy is a subtype of gaze palsy in which conjugate, horizontal eye movements are limited by neurologic deficits. Horizontal gaze palsies typically result from an ipsilateral pontine lesion or a contralateral frontal lobe lesion. [1]
Babies suffering from PTU may exhibit normal or slightly jerky side-to-side eye movement, nausea, irritability, frequent sleep, developmental and language delays, vertigo and loss of muscle tone. [citation needed] The condition is generally regarded as having a benign outcome, in the sense that it improves, rather than worsens over time.