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Aspirin works to help reduce the appearance of keloid scars by preventing the cells that form the scars from entering the wound site in the first place. To do this, you can crush approximately ...
A keloid scar is benign and not contagious, but sometimes accompanied by severe itchiness, pain, [2] and changes in texture. In severe cases, it can affect movement of skin. In the United States, keloid scars are seen 15 times more frequently in people of sub-Saharan African descent than in people of European descent. [3]
Lysine acetylsalicylate, also known as aspirin DL-lysine or lysine aspirin, is a more soluble form of acetylsalicylic acid (aspirin). As with aspirin itself, it is a nonsteroidal anti-inflammatory drug (NSAID) with analgesic, anti-inflammatory, antithrombotic and antipyretic properties. [ 1 ]
A hypertrophic scar is a cutaneous condition characterized by deposits of excessive amounts of collagen which gives rise to a raised scar, but not to the degree observed with keloids. [1] Like keloids, they form most often at the sites of pimples, body piercings, cuts and burns. They often contain nerves and blood vessels.
Scarification can also help change status from victim to survivor. These individuals pass through various kinds of ritual death and rebirth, and redefine the relationship between self and society through the skin. [7] Many people in certain regions of Africa who have "markings" can be identified as belonging to a specific tribe or ethnic group.
Keloids can also form inside the body. After spinal surgery, a Keloid can form around the nerve roots, leading to "failed back surgery" syndrome. The Keloid can continue to grow for as long as 18 months post-operatively. In some cases, the scar tissue is so dense that the nerve roots can no longer be observed in an MRI.
Aspirin is non-selective and irreversibly inhibits both forms [4] (but is weakly more selective for COX-1 [5]). It does so by acetylating the hydroxyl of a serine residue at the 530 amino acid position. [6] Normally COX produces prostaglandins, most of which are pro-inflammatory, and thromboxanes, which promote clotting.
The exact mechanism of action of silicone gel sheeting has not been fully studied. Currently, many proposed mechanisms explain the efficacy of such treatment, including the occlusion and hydration effect, increased body surface temperature, polarized electric charge, immunological effects, etc. [9] The occlusion and hydration effect is the most studied mechanism of action.