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Thrombocytopenia affects approximately 7–10% of pregnant women and of the 7–10%, within that population; approximately 70–80% have gestational thrombocytopenia [3] Gestational thrombocytopenia is a disorder similar to immune thrombocytopenia (ITP) and is difficult to differentiate between the two disorders. [ 2 ]
Unlike hemolytic disease of the fetus and newborn, NAIT occurs during the first pregnancy in up to 50% of cases, [1] and the affected fetuses may develop severe thrombocytopenia (<50,000 μL −1) very early during pregnancy (as early as 20 weeks gestation, consistent with the development of platelet antigens, [1] and the majority of the time ...
ITP can be difficult to distinguish from gestational thrombocytopenia (which is by far the most common cause of thrombocytopenia in pregnancy). Unlike ITP, the platelet count in gestational thrombocytopenia rarely goes below 100,000, and a platelet count below 80,000 is even more rare (seen in less than 0.1% of cases of gestational ...
645.1 Post term pregnancy; 645.2 Prolonged pregnancy; 646 Other complications of pregnancy, not elsewhere classified 646.0 Papyraceous fetus; 646.1 Edema or excessive weight gain in pregnancy without mention of hypertension; 646.2 Unspecified renal disease in pregnancy without mention of hypertension; 646.3 Habitual aborter currently pregnant
The ISTH guidelines [51] recommended for first acute episode and relapses of immune-mediated TTP (iTTP), add corticosteroids to therapeutic plasma exchange (TPE) and consider adding rituximab and caplacizumab. For asymptomatic iTTP with low plasma ADAMTS13 activity, consider rituximab outside of pregnancy, but prophylactic TPE during pregnancy ...
Pregnancy-induced hypercoagulability is probably a physiologically adaptive mechanism to prevent post partum hemorrhage. [1] Pregnancy changes the plasma levels of many clotting factors, such as fibrinogen, which can rise up to three times its normal value. [2] Thrombin levels increase. [3] Protein S, an anticoagulant, decreases.