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There are several sets of criteria used to diagnose LVH via electrocardiography. [10] None of them are perfect, though by using multiple criteria sets, the sensitivity and specificity are increased. The Sokolow-Lyon index: [11] [12] S in V 1 + R in V 5 or V 6 (whichever is larger) ≥ 35 mm (≥ 7 large squares) R in aVL ≥ 11 mm
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LVH may interfere with heart functionality in a number of ways. Before progression to a dilated phenotype, mechanical obstruction of the outflow tract can occur, leading to reduced cardiac output. Additionally, increased fibrosis of the ventricle can result in a failure to relax appropriately which impairs cardiac filling and may lead to ...
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With the absence of monocytes and macrophages, chances of LVT formation are very high. Failure to clear cellular debris from the infarct compromises the endothelial lining of the left ventricle and exposes the damaged tissue to the blood. [6] The response is to build a thrombus composed of fibrin, red blood cells and platelets. [1]
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Ventricular aneurysms usually grow at a very slow pace, but can still pose problems. Usually, this type of aneurysm grows in the left ventricle.This bubble has the potential to block blood flow to the rest of the body, and thus limit the patient's stamina.
Example ultrasound of an athlete. Athlete's heart most often does not have any physical symptoms, although an indicator would be a consistently low resting heart rate.. Athletes with AHS often do not realize they have the condition unless they undergo specific medical tests, because athlete's heart is a normal, physiological adaptation of the body to the stresses of physical conditioning and ...