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440.1 Stenosis of renal artery; 440.2 Peripheral Arterial Disease 440.21 Peripheral Arterial Disease with Intermittent Claudication (Also Claudication) 440.23 Peripheral Arterial Disease w/ ulceration; 441 Aortic aneurysm and dissection. 441.0 Aortic Dissection; 441.3 Abdominal Aortic Aneurysm, ruptured; 441.4 Abdominal aortic Aneurysm, w/o rupture
Renal artery stenosis (RAS) is the narrowing of one or both of the renal arteries, most often caused by atherosclerosis or fibromuscular dysplasia. This narrowing of the renal artery can impede blood flow to the target kidney , resulting in renovascular hypertension – a secondary type of high blood pressure .
Radioisotopes can differentiate between passive dilatation and obstruction. It is widely used before kidney transplantation to assess the vascularity of the kidney to be transplanted and with a test dose of captopril to highlight possible renal artery stenosis in the donor's other kidney, [12] and later the performance of the transplant.
Renal infarction is a medical condition caused by an abrupt disruption of the renal blood flow in either one of the segmental branches or the major ipsilateral renal artery. [3] Patients who have experienced an acute renal infarction usually report sudden onset flank pain , which is often accompanied by fever , nausea , and vomiting .
This leads to a build-up of plaques and they can be deposited in the renal arteries causing stenosis and ischemic kidney disease. [citation needed] In this situation, the kidney supplied blood by the narrowed renal artery suffers from inadequate blood flow, which in turn causes the size of the kidneys to decrease. Other consequences include ...
The procedure involves endovascular access via the femoral artery with advancement of a catheter-mounted device into the renal artery. The device uses radiofrequency or ultrasound to ablate the renal nerves. Typically, numerous ablations are applied at a different longitudinal and rotational positions to ensure maximal denervation. [13]
Angiotension converting enzyme. The pathogenesis of renovascular hypertension involves the narrowing of the arteries supplying the kidneys which causes a low perfusion pressure that is detected by the juxtaglomerular apparatus (via the macula densa cells, which act as baroreceptors; located on the afferent arteriole wall). [6]
Atherosclerotic obstruction of the renal artery can be treated with angioplasty with or without stenting of the renal artery. [10] There is a weak recommendation for renal artery angioplasty in patients with renal artery stenosis and flash edema or congestive heart failure. [10]