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Myotonia congenita is a congenital neuromuscular channelopathy that affects skeletal muscles (muscles used for movement). It is a genetic disorder.The hallmark of the disease is the failure of initiated contraction to terminate, often referred to as delayed relaxation of the muscles and rigidity. [1]
Drug induced tremor G25.1 Other specified form of tremor G25.2 Myoclonus: 333.2 G25.3 Chorea (rapid, involuntary movement) Drug induced chorea: G25.4 Drug-induced tics and tics of organic origin 333.3 G25.6 Paroxysmal nocturnal limb movement G25.80 Painful legs (or arms), moving toes (or fingers) syndrome G25.81 Sporadic restless leg syndrome ...
In human sexuality, paralysis, also known as rape paralysis, [1] involuntary paralysis, [2] fright (or faint), [3] [4] or tonic immobility, [1] [3] [5] is a natural bodily survival reaction which can be automatically activated by the brain of a person who feels threatened by sexual violence. During this paralysis, one cannot move and cannot say ...
The term cataplexy originates from the Greek κατά (kata, meaning "down"), and πλῆξις (plēxis, meaning "strike") [4] and it was first used around 1880 in German physiology literature to describe the phenomenon of tonic immobility also known as "playing possum" (in reference to the opossum's behavior of feigning death when threatened ...
Drugs that interfere with nerve function, such as curare, can also cause paralysis. Pseudoparalysis ( pseudo- meaning "false, not genuine", from Greek ψεῦδος [ 7 ] ) is voluntary restriction or inhibition of motion because of pain, incoordination, orgasm, or other cause, and is not due to actual muscular paralysis. [ 8 ]
Dystonia is often intensified or exacerbated by physical activity, and symptoms may progress into adjacent muscles. [ 4 ] The disorder may be hereditary or caused by other factors such as birth-related or other physical trauma , infection , poisoning (e.g., lead poisoning ) or reaction to pharmaceutical drugs , particularly neuroleptics , [ 3 ...
The antimalarial drug quinine is a traditional treatment that may be slightly effective for reducing the number of cramps, the intensity of cramps, and the number of days a person experiences cramps. Quinine has not been shown to reduce the duration (length) of a muscle cramp. [6] Quinine treatment may lead to haematologic and cardiac toxicity.
In the past, dopamine blocking agents have been used in the treatment of spasmodic torticollis. Treatment was based on the theory that there is an imbalance of the neurotransmitter dopamine in the basal ganglia. These drugs have fallen out of fashion due to various serious side effects: sedation, parkinsonism, and tardive dyskinesia. [16]