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In contrast to the cell-dependent model, the cell-autonomous model proposes that HIV latency decisions are largely driven by the Tat-positive feedback loop and latency is therefore a probabilistic response due to intrinsically generated phenotypic heterogeneity rather than host-cell-determined. [12]
Furthermore, the PARTNER study, [43] which ran from 2010 to 2014, enrolled 1166 serodiscordant couples (where one partner is HIV positive and the other is negative) in a study that found that the estimated rate of transmission through any condomless sex with the HIV-positive partner taking ART with an HIV load less than 200 copies/ml was zero. [43]
HIV is commonly transmitted via unprotected sexual activity, blood transfusions, hypodermic needles, and from mother to child. Upon acquisition of the virus, the virus replicates inside and kills T helper cells , which are required for almost all adaptive immune responses .
Contracting HIV can lead to the development of AIDS or stage 3 HIV, which causes serious damage to the immune system. [medical citation needed] While this virus is the underlying cause of AIDS, [3] not all HIV-positive individuals have AIDS, as HIV can remain in a latent state for many years.
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The latest recommendations of the US Centers for Disease Control and Prevention (CDC) show that HIV testing must start with an immunoassay combination test for HIV-1 and HIV-2 antibodies and p24 antigen. A negative result rules out HIV exposure, while a positive one must be followed by an HIV-1/2 antibody differentiation immunoassay to detect ...
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