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Profound abnormalities detected with visual field and multifocal electroretinography testing can be observed in the presence of a normal retinal appearance. Retinal examinations are advised for documentation, but visible bull's-eye maculopathy is a late change, [6] and the goal of screening is to recognize toxicity at an earlier stage. Annual ...
Toxic retinopathies, including those caused by Plaquenil and Vigabatrin. The ERG is also used to monitor retinal toxicity in many drug trials. Autoimmune retinopathies such as cancer associated retinopathy (CAR), melanoma associated retinopathy (MAR), and acute zonal occult outer retinopathy (AZOOR) Retinal detachment
There are several causes of toxic optic neuropathy. [1] Among these are: ingestion of methanol (wood alcohol), ethylene glycol (automotive antifreeze), disulfiram (used to treat chronic alcoholism), halogenated hydroquinolones (amebicidal medications), ethambutol and isoniazid (tuberculosis treatment), and antibiotics such as linezolid and chloramphenicol as well as chloroquine and the related ...
Macular toxicity is related to the total cumulative dose rather than the daily dose. Regular eye screening, even in the absence of visual symptoms, is recommended to begin when either of these risk factors occurs. [22] Toxicity from hydroxychloroquine may be seen in two distinct areas of the eye: the cornea and the macula.
Confrontation visual field testing is an important part of a routine ophthalmological or neurological examination. It can be used for rapid and gross assessment of large-scale visual field problems due to ophthalmological or neurological diseases, such as homonymous and heteronymous hemianopias, quadranopsia, altitudinal visual loss, central/centrocecal scotoma etc. [1] [2] Test using a red ...
LHON usually presents with rapid vision loss in one eye followed by involvement of the second eye (usually within months). Visual acuity often remains stable and poor (around or below 20/200) with a residual central visual field defect. Patients with the 14484/ND6 mutation are most likely to have visual recovery. [8]
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This visual field defect is called as bitemporal hemianopia. Anterior chiasmal syndrome, the lesions that affect the ipsilateral optic nerve fibres and the contralateral inferonasal fibres located in the Willebrand knee produce junctional scotoma, i.e., a combination of central scotoma in one eye and temporal hemianopia defect in the other eye. [1]