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The most illustrative effects of Agent Orange upon the Vietnamese people are the health effects. [4] Scientific consensus has made it clear that the importance of accuracy in terms of site-specific cancer risk as well as the difficulty in identifying Agent Orange as the cause of that specific cancer risk must be acknowledged.
In 2011, an appraisal of the 20-year long Air Force Health Study that began in 1982 indicates that the results of the AFHS as they pertain to Agent Orange, do not provide evidence of disease in the Operation Ranch Hand veterans caused by "their elevated levels of exposure to Agent Orange".
Agent Orange Act of 1991; Long title: An Act to provide for the Secretary of Veterans Affairs to obtain independent scientific review of the available scientific evidence regarding associations between diseases and exposure to dioxin and other chemical compounds in herbicides, and for other purposes.
Agent Orange III: 66.6% n-butyl 2,4-D and 33.3% n-butyl ester 2,4,5-T. [12] Enhanced Agent Orange, Orange Plus, Super Orange (SO), or DOW Herbicide M-3393: standardized Agent Orange mixture of 2,4-D and 2,4,5-T combined with an oil-based mixture of picloram, a proprietary Dow Chemical product called Tordon 101, an ingredient of Agent White. [13 ...
Officials caution that individuals who were inside Tom Bradley International Terminal B at LAX from about 2:30 to 4 p.m. Friday may be at risk of developing measles.
The substances that may cause chloracne are now collectively known as chloracnegens. Chloracne is particularly linked to toxic exposure to dioxins (byproducts of many chemical processes, including the manufacture of herbicides such as Agent Orange)—so much so that it is considered a
Agent Orange, a defoliant used by the British in the Malayan Emergency and the U.S. in the Vietnam War, was equal parts 2,4,5-T and 2,4-D (2,4-dichlorophenoxyacetic acid). 2,4,5-T itself is toxic with a NOAEL of 3 mg/kg/day and a LOAEL of 10 mg/kg/day. [3] Agent Pink contained 100% 2,4,5-T (dioxin
Causes: Exposure to acetylcholinesterase inhibitors such as sarin, and possibly other toxic exposures during the 1990–91 Persian Gulf War. See Congressional records for testimony about chemical weapon disposal accident. Risk factors: Lower levels of PON1, a protein in humans responsible for breaking down organophosphates. Differential diagnosis