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All patients showed cerebellar and/or vermal dysfunction and, on formal psychometric testing, cognitive abilities ranged from normal to moderately retarded. Abnormalities on CT scan ranged from prominent valleculla to an enlarged cisterna magna with hypoplasia of the cerebellar hemispheres and vermis.
The cisterna magna (posterior cerebellomedullary cistern, [1] or cerebellomedullary cistern [2] [3]) is the largest of the subarachnoid cisterns.It occupies the space created by the angle between the caudal/inferior surface of the cerebellum, and the dorsal/posterior surface of the medulla oblongata (it is created by the arachnoidea that bridges this angle [3]).
Mega cisterna magna is a condition in which the cisterna magna, the subarachnoid cistern below the fourth ventricle, is enlarged. It has been proposed to be due to a delayed rupture of Blake's pouch rather than a failed rupture. [9] In mega cisterna magna, unlike in DWM: [9] The cerebellum is not usually hypoplastic.
The quadrigeminal cistern [1] (also cistern of great cerebral vein, [1] vein of Galen cistern, [2] superior cistern, [2] [3] Bichat's canal, [3] or peripineal cistern [2]) is a subarachnoid cistern situated between splenium of corpus callosum, and the superior surface of the cerebellum.
The cistern of lateral cerebral fossa [1] (also cistern of the lateral sulcus, or Sylvian cistern [2]) is an elongated [3] subarachnoid cistern formed by arachnoid mater bridging the lateral sulcus between the frontal, temporal, and parietal opercula.
Agenesis of the corpus callosum (ACC) is a rare birth defect in which there is a complete or partial absence of the corpus callosum.It occurs when the development of the corpus callosum, the band of white matter connecting the two hemispheres in the brain, in the embryo is disrupted.
Fourth ventricle location shown in red (E), pons (B); the floor of the ventricle is to the right, the roof to the left. The fourth ventricle has a roof at its upper (posterior) surface and a floor at its lower (anterior) surface, and side walls formed by the cerebellar peduncles (nerve bundles joining the structure on the posterior side of the ventricle to the structures on the anterior side).
Tornwaldt's disease is a rare benign disorder caused by persistent notochord remnants. [3] This disease almost remains asymptomatic. [citation needed] At about the 10th week of embryonic development, the pharyngeal pouch forms by adhesion of the pharyngeal ectoderm to the cranial end of the notochord.