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Prosopagnosia, [2] also known as face blindness, [3] is a cognitive disorder of face perception in which the ability to recognize familiar faces, including one's own face (self-recognition), is impaired, while other aspects of visual processing (e.g., object discrimination) and intellectual functioning (e.g., decision-making) remain intact.
Congenital prosopagnosia is an inability to recognize faces without a history of brain damage; while acquired prosopagnosia is caused by damage to the right occipital-temporal region of the brain. In the 1950s it was theorized that the right cerebral hemisphere was involved in facial recognition and in the 1960s this theory was supported by ...
Prosopagnosia (inability to recognize faces) is due to damage of the fusiform face area (FFA). An area in the fusiform gyrus of the temporal lobe that has been strongly associated with a role in facial recognition. [6] However, this area is not exclusive to faces; recognition of other objects of expertise are also processed in this area.
Bruce & Young Model of Face Recognition, 1986. One of the most widely accepted theories of face perception argues that understanding faces involves several stages: [7] from basic perceptual manipulations on the sensory information to derive details about the person (such as age, gender or attractiveness), to being able to recall meaningful details such as their name and any relevant past ...
A specific form of associative visual agnosia is known as prosopagnosia. Prosopagnosia is the inability to recognize faces. For example, these individuals have difficulty recognizing friends, family and coworkers. [22] However, individuals with prosopagnosia can recognize all other types of visual stimuli. [23]
"Several studies have indicated that as many as 1 in 50 people may have developmental prosopagnosia," the NHS reports, meaning as many as 6.5 million people in the U.S. could have it, to varying ...
When those with prosopagnosia view faces, the fusiform gyrus (a facial recognition area of the brain) activates differently to how it would in someone without the condition. [27] Additionally, non-facial object recognition areas (such as the ventral occipitotemporal extrastriate cortex ) are activated when viewing faces, suggesting that faces ...
In this way, it is very easily mistaken as prosopagnosia, which is an inability to perceive or recognize faces. Prosopagnosia is a deficit that occurs earlier in the neural circuit while the facial stimuli is being processed, whereas prosopamnesia takes effect when the brain attempts to encode the processed facial stimuli into memory.