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Neuroplasticity is the process by which neurons adapt to a disturbance over time, and most often occurs in response to repeated exposure to stimuli. [27] Aerobic exercise increases the production of neurotrophic factors [note 1] (e.g., BDNF, IGF-1, VEGF) which mediate improvements in cognitive functions and various forms of memory by promoting blood vessel formation in the brain, adult ...
Bromocriptine is a partial agonist of the dopamine D 2 receptor. [21] [22] [23] It also interacts with other dopamine receptors and with various serotonin and adrenergic receptors. [21] [22] [24] Bromocriptine has additionally been found to inhibit the release of glutamate by reversing the GLT1 glutamate transporter. [25]
l-DOPA is produced from the amino acid l-tyrosine by the enzyme tyrosine hydroxylase. l-DOPA can act as an l-tyrosine mimetic and be incorporated into proteins by mammalian cells in place of l-tyrosine, generating protease-resistant and aggregate-prone proteins in vitro and may contribute to neurotoxicity with chronic l-DOPA administration. [10]
Dopamine therapy is the regulation of levels of the neurotransmitter dopamine through the use of either agonists, or antagonists; and has been used in the treatment of disorders characterized by a dopamine imbalance. Dopamine replacement therapy (DRT) is an effective treatment for patients with decreased levels of dopamine.
It has an EC 50 Tooltip half-maximal effective concentration for dopamine release of 48.7 nM but induces only 85% release of norepinephrine at a concentration of 10 μM. [16] For comparison, the EC 50 values of the NDRA methcathinone are 49.9 nM for dopamine release and 22.4 nM for norepinephrine release and it induces 100% release of ...
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