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[8] [9] CIDP is extremely rare but under-recognized and under-treated due to its heterogeneous presentation (both clinical and electrophysiological) and the limitations of clinical, serologic, and electrophysiologic diagnostic criteria. Despite these limitations, early diagnosis and treatment is favoured in preventing irreversible axonal loss ...
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The rate of incidence of alcoholic polyneuropathy involving sensory and motor polyneuropathy has been stated as from 10% to 50% of alcoholics depending on the subject selection and diagnostic criteria. If electrodiagnostic criteria are used, alcoholic polyneuropathy may be found in up to 90% of individuals being assessed. [4]
Cramp fasciculation syndrome is diagnosed by clinical examination and electromyography (EMG). [5] Fasciculation is the only abnormality [6] (if any) [7] seen with EMG. Cramp fasciculation syndrome is a chronic condition. [8] Treatment options include anti-seizure medications such as carbamazepine, immunosuppressive drugs and plasmapheresis.
Needle EMG is used as a diagnostics tool for identifying neuromuscular diseases, [5] or as a research tool for studying kinesiology, and disorders of motor control. EMG signals are sometimes used to guide botulinum toxin or phenol injections into muscles. Surface EMG is used for functional diagnosis and during instrumental motion analysis.
The current diagnosis criteria for MS do not allow doctors to give an MS diagnosis until a second attack takes place. Therefore, the concept of "clinical MS", for an MS that can be diagnosed, has been developed. Until MS diagnosis has been established, nobody can tell whether the disease one is dealing with is MS. [citation needed]
Collaborators do not perform or interpret needle electromyography (EMG) studies or interpret NCSs but are active in the field of neuromuscular medicine. AANEM research members are currently active in neuromuscular or electrodiagnostic research and are a PhD investigator, engineer, holder of a master's degree, or graduate student enrolled in a ...
His research began in 1948, with the application of EMG to myasthenia gravis. [4] In 1956, he and colleague Lee Eaton were the first to describe the clinical and EMG characteristics of the disease now known as Lambert–Eaton myasthenic syndrome. This was the first pre-synaptic disease found, differentiating it from myasthenia gravis. [10]