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Itopride poorly penetrates across the blood brain barrier because of its high polarity and thus itopride does not tend to cause any central nervous system adverse effects. [7] Itopride has no effect on potassium channels. [15] Similarly to other D 2 receptor antagonists, itopride has been found to dose-dependently increase prolactin levels. [6]
Hennessey Performance Engineering (HPE) is an American hypercar manufacturer and high-performance vehicle creator. In addition to building the Venom F5 hypercar, the company specializes in 'making fast cars faster' modifying sports cars from several brands including Chevrolet , Dodge , Cadillac , Jeep , [ 1 ] Ford , GMC , and Lincoln .
Pages in category "Automotive motorsports and performance companies" The following 157 pages are in this category, out of 157 total. This list may not reflect recent changes .
In 2005, it became the sole owner of Ilmor and renamed the company first to Mercedes-Benz High Performance Engines, then to Mercedes-Benz HighPerformanceEngines. In December 2011, the company was renamed to Mercedes AMG High Performance Powertrains along with the renaming of Mercedes GP to incorporate the Mercedes-AMG sub-brand.
The 2005 season was similarly successful as the company took its second Indy 500 victory and won 12 races to win the manufacturers' and drivers' crowns. [9] From 2006 to 2011 , HPD was the sole engine manufacturer of the IndyCar Series, including the Indianapolis 500.
Tiapride is a drug that selectively blocks D 2 and D 3 dopamine receptors in the brain. It is used to treat a variety of neurological and psychiatric disorders including dyskinesia, alcohol withdrawal syndrome, negative symptoms of psychosis, and agitation and aggression in the elderly. [2]
Ford Performance (formerly Ford Racing) is the high-performance division of the Ford Motor Company and the multinational name used for its motorsport and racing activity. History [ edit ]
Amisulpride is approved and used at low doses in the treatment of dysthymia and major depressive disorder. [10] [20] [11] [21] [22] [23] Whereas typical doses used in schizophrenia block postsynaptic dopamine D 2-like receptors and reduce dopaminergic neurotransmission, low doses of amisulpride preferentially block presynaptic dopamine D 2 and D 3 autoreceptors and thereby disinhibit dopamine ...