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Recovery from a reflex syncope episode happens without specific treatment. [2] Prevention of episodes involves avoiding a person's triggers. [2] Drinking sufficient fluids, salt, and exercise may also be useful. [2] [4] If this is insufficient for treating vasovagal syncope, medications such as midodrine or fludrocortisone may be tried. [4]
Radiation as a treatment for malignant head and neck tumors can reduce the number of blood vessels in the treatment area and lead to scarring. In some cases, this can paralyze the vagus nerve, of which the RLNs are branches. [9] Tumors of the vagus nerve, called vagal neurilemmomas, can also paralyze the vocal folds. [9]
The vagus nerve is also responsible for regulating inflammation in the body, via the inflammatory reflex. [7] Efferent vagus nerve fibers innervating the pharynx and back of the throat are responsible for the gag reflex. In addition, 5-HT 3 receptor-mediated afferent vagus stimulation in the gut due to gastroenteritis is a cause of vomiting. [8]
The inferior pharyngeal constrictor muscle is a skeletal muscle of the neck. It is the thickest of the three outer pharyngeal muscles. It arises from the sides of the cricoid cartilage and the thyroid cartilage. It is supplied by the vagus nerve (CN X). It is active during swallowing, and partially during breathing and speech.
Fibers cross over to and join the vagus nerve in the jugular foramen. [8]: 86–88 Sensory cell bodies are located in the inferior jugular ganglion, [9] and the fibers terminate in the solitary nucleus. [8]: 86–88 Parasympathetic fibers to segments of the trachea and esophagus in the neck originate in the dorsal nucleus of the vagus nerve. [9]
Vagus nerve stimulation can also increase the release of important brain chemicals such as serotonin and norepinephrine, which help regulate mood and are typically low in people with depression ...