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Primary and secondary brain injury are ways to classify the injury processes that occur in brain injury. In traumatic brain injury (TBI), primary brain injury occurs during the initial insult, and results from displacement of the physical structures of the brain. [1] Secondary brain injury occurs gradually and may involve an array of cellular ...
Cerebral achromatopsia occurs after injury to the lingual or fusiform gyrus, the areas associated with hV4. These injuries include physical trauma, stroke, and tumour growth. One of the primary initiatives to locating the colour centre in the visual cortex is to discover the cause and a possible treatment of cerebral achromatopsia.
Vehicle accidents are the most frequent cause of DAI; it can also occur as the result of child abuse [15] such as in shaken baby syndrome. [16] Immediate disconnection of axons may be observed in severe brain injury, but the major damage of DAI is delayed secondary axon disconnections, slowly developed over an extended time course. [2]
Primary brain injury (the damage that occurs at the moment of trauma when tissues and blood vessels are stretched, compressed, and torn) is not adequate to explain this deterioration; rather, it is caused by secondary injury, a complex set of cellular processes and biochemical cascades that occur in the minutes to days following the trauma. [73]
Diffuse axonal injury is caused by shearing forces on the brain leading to lesions in the white matter tracts of the brain. [31] These shearing forces are seen in cases where the brain had a sharp rotational acceleration, and is caused by the difference in density between white matter and grey matter.
Neuromelanin gives specific brain sections, such as the substantia nigra or the locus coeruleus, distinct color. It is a type of melanin and similar to other forms of peripheral melanin. It is insoluble in organic compounds, and can be labeled by silver staining. It is called neuromelanin because of its function and the color change that ...
Cerebral achromatopsia is a type of color blindness caused by damage to the cerebral cortex of the brain, rather than abnormalities in the cells of the eye's retina.It is often confused with congenital achromatopsia but the underlying physiological deficits of the disorders are completely distinct.
The colors in each pair oppose each other. Red-green receptors cannot send messages about both colors at the same time. This theory also explains negative afterimages; once a stimulus of a certain color is presented, the opponent color is perceived after the stimulus is removed because the anabolic and catabolic processes are reversed. For ...