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The difference in memory between normal aging and a memory disorder is the amount of beta-amyloid deposits, hippocampal neurofibrillary tangles, or amyloid plaques in the cortex. If there is an increased amount, memory connections become blocked, memory functions decrease much more than what is normal for that age and a memory disorder is ...
The memory is stored in long-term memory, but access to it is impaired because of psychological defense mechanisms. Persons retain the capacity to learn new information and there may be some later partial or complete recovery of memory. Formerly known as "Psychogenic amnesia".
Long-term memory (LTM) is the stage of the Atkinson–Shiffrin memory model in which informative knowledge is held indefinitely. It is defined in contrast to sensory memory, the initial stage, and short-term or working memory, the second stage, which persists for about 18 to 30 seconds.
Memory lapses like these are common for people of all ages. “Mild forgetfulness — you forget somebody’s name or where you left something — that’s totally normal,” says Karlene Ball, Ph.D.
McGaugh and colleagues posit that although electrical and pharmaceutical stimulation directly to the amygdala can enhance or decrease memory, the amygdala is not the main site for any long-term memory storage. Rather, the amygdala acts as a modulator for storage processes occurring in other areas of the brain.
Difficulty creating recent term memories is called anterograde amnesia and is caused by damage to the hippocampus part of the brain, which is a major part of the memory process. [8] Retrograde amnesia is also caused by damage to the hippocampus, but the memories that were encoded or in the process of being encoded in long-term memory are erased [8]