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The response to treatment is measured by sustained viral response (SVR), defined as the absence of detectable RNA of the hepatitis C virus in blood serum for at least 24 weeks after discontinuing treatment, [112] and rapid virological response (RVR), defined as undetectable levels achieved within four weeks of treatment. Successful treatment ...
However, since hepatitis C is known to have been present since at least the 1940s, a gamma globulin shot received prior to the early 1990s put the recipient at risk of being infected. Intravenous gamma globulin was FDA-approved in 2004 to reduce antibodies in a patient with kidney failure to allow that person to accept a kidney from a donor ...
Microsomal antibody Antigen Disease anti-LKM 1: cytochrome P450 2D6: autoimmune hepatitis type II and chronic hepatitis C (10%) anti-LKM 2: cytochrome P450 2C9: drug-induced hepatitis (tienilic acid–induced) anti-LKM 3: cytochrome P450 1A2: chronic active hepatitis in association with autoimmune polyendocrine syndrome type 1; [1] hepatitis D
The hepatitis C virus (HCV) [3] is a small (55–65 nm in size), enveloped, positive-sense single-stranded RNA virus of the family Flaviviridae. The hepatitis C virus is the cause of hepatitis C and some cancers such as liver cancer ( hepatocellular carcinoma , abbreviated HCC) and lymphomas in humans.
The presence of surface antibody (anti-HBs) indicates an individual with immunity to hepatitis B, whether due to previously resolved infection or due to hepatitis B vaccination. [65] For example, an individual who has never had any exposure to HBV, either by vaccine or by infection, would test negative for the entire serology panel.
hepatitis B surfage antigen: hepatitis B Lenzilumab [6] mab: human: CSF2: chronic myelomonocytic leukemia and juvenile myelomonocytic leukemia: Lerdelimumab [17] mab: human: TGF-β2: reduction of scarring after glaucoma surgery: Leronlimab [5] mab: humanized: CCR5: breast cancer, HIV Lesofavumab [28] mab: human: Hemagglutinin (influenza ...
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