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Purkinje cells of the cerebellar cortex tonically inhibit deep nuclear cells. Therefore, an LTD-mediated decrease in PC activity at the appropriate time during a CS-US interval could release the INP from tonic inhibition and allow for execution of a CR. An increase in PC activity could have the opposite effect, prohibiting or limiting CR execution.
In humans, the cerebrum is the largest and best-developed of the five major divisions of the brain. The cerebrum is made up of the two cerebral hemispheres and their cerebral cortices (the outer layers of grey matter), and the underlying regions of white matter. [2] Its subcortical structures include the hippocampus, basal ganglia and olfactory ...
In neuroscience, cortical magnification describes how many neurons in an area of the visual cortex are 'responsible' for processing a stimulus of a given size, as a function of visual field location. [ a ] In the center of the visual field, corresponding to the center of the fovea of the retina , a very large number of neurons process ...
The outer part of the cerebrum is the cerebral cortex, made up of grey matter arranged in layers. It is 2 to 4 millimetres (0.079 to 0.157 in) thick, and deeply folded to give a convoluted appearance. [21] Beneath the cortex is the cerebral white matter. The largest part of the cerebral cortex is the neocortex, which has
Lesions to this area can result in multiple deficits in visual tracking and oculomotor control (such as nystagmus and vertigo), integration of vestibular information for eye and head control, as well as control of axial muscles for balance. [2] The most common cause of damage to the flocculonodular lobe is medulloblastoma in childhood ...
The cerebellar peduncles are three paired bundles of fibres that connect the cerebellum to the brain stem. [1]Superior cerebellar peduncle is a paired structure of white matter that connects the cerebellum to the mid-brain.
The frontal eye fields (FEF) are a region located in the frontal cortex, more specifically in Brodmann area 8 or BA8, [1] of the primate brain. In humans, it can be more accurately said to lie in a region around the intersection of the middle frontal gyrus with the precentral gyrus , consisting of a frontal and parietal portion. [ 2 ]
The pathway then continues out of the eye to the layers in-between the parvocellular and magnocellular layers of the dLGN. This pathway then terminates at the blobs in V1. [2] Lesioning of the koniocellular pathway leads to lack of acuity in shapes and colour.