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The diathesis-stress model, also known as the vulnerability–stress model, is a psychological theory that attempts to explain a disorder, or its trajectory, as the result of an interaction between a predispositional vulnerability, the diathesis, and stress caused by life experiences.
The idea that individuals vary in their sensitivity to their environment was historically framed in diathesis-stress [4] or dual-risk terms. [5] These theories suggested that some "vulnerable" individuals, due to their biological, temperamental and/or physiological characteristics (i.e., "diathesis" or "risk 1"), are more vulnerable to the adverse effects of negative experiences (i.e., "stress ...
The concept of Environmental Sensitivity integrates multiple theories on how people respond to negative and positive experiences. These include the frameworks of Diathesis-stress model [4] and Vantage Sensitivity, [5] as well as the three leading theories on more general sensitivity: Differential Susceptibility, [6] [7] Biological Sensitivity to Context, [8] and Sensory processing sensitivity ...
A related view, the diathesis-stress model, posits that mental disorders result from genetic dispositions and environmental stressors, combining to cause patterns of distress or dysfunction. [19] The model is one way to explain why some individuals are more vulnerable to mental disorders than others.
The diathesis-stress model is an interactionism approach. In the context of schizophrenia, diathesis is the vulnerability. Vulnerabilities can be either genetic predisposition or early experiences, or both. [22] Stressors are any event that can trigger a schizophrenia-vulnerable individual to the onset of the condition.
The diathesis–stress model [23] emphasizes the importance of applying multiple causality to psychopathology, by stressing that disorders are caused by both precipitating causes, and predisposing causes. A precipitating cause is an immediate trigger that instigates a person's action or behavior.
[5] [37] The diathesis–stress model specifies that depression results when a preexisting vulnerability, or diathesis, is activated by stressful life events. The preexisting vulnerability can be either genetic , [ 38 ] [ 39 ] implying an interaction between nature and nurture , or schematic , resulting from views of the world learned in ...
The investigators suggested that in utero insults may specifically stress those with a schizophrenia diathesis, supporting a "diathesis-stress" etiological model for a subset of schizophrenia (that Kraepelin identified) with early abnormalities suggesting brain atrophy. [57]