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Applying auxin to the cut stem again inhibits lateral dominance. [2] Moreover, it has been shown that cytokinin alone has no effect on parenchyma cells. When cultured with auxin but no cytokinin, they grow large but do not divide. When cytokinin and auxin are both added together, the cells expand and differentiate.
R.fascians can also degrade cytokinin to influence the cytokinin/auxin ratio. [citation needed] Beside cytokinin and auxin, R. fascians acts on other hormones: in particular, it can block abscisic acid and gibberellic acid synthesis in infected plants. Abscisic acid represses growth, so a block of production is needed to allow proliferation of ...
The most important member of the auxin family is indole-3-acetic acid (IAA), [7] which generates the majority of auxin effects in intact plants, and is the most potent native auxin. And as native auxin, its equilibrium is controlled in many ways in plants, from synthesis, through possible conjugation to degradation of its molecules, always ...
Zeatin, a cytokinin Cytokinins (CKs) are a group of chemicals that influence cell division and shoot formation. They also help delay senescence of tissues, are responsible for mediating auxin transport throughout the plant, and affect internodal length and leaf growth.
Experiments have reported that application of AHLs activates the auxin-responsible GH3 promoter (upregulate auxin-related genes), and down-regulates the genes related to cytokinin (the change in the ratio between auxin and cytokinin can promote growth). [16] Also, followed by applying AHL, the nodulation in roots has been enhanced. [17]
Kinetin is often used in plant tissue culture to induce callus formation (in conjunction with auxin) and regenerate shoot tissues from callus (with lower auxin concentration). For a long time, it was believed that kinetin was an artifact produced from the deoxyadenosine residues in DNA , which degraded when standing for long periods or when ...
The auxin indole-3-acetic acid (IAA) regulates concentration of GA 1 in elongating internodes in peas. [37] Removal of IAA by removal of the apical bud, the auxin source, reduces the concentration of GA 1, and reintroduction of IAA reverses these effects to increase the concentration of GA 1. [37] This has also been observed in tobacco plants. [38]
Within the 20-year timespan, many scientists have actively contributed to examining and reevaluating Hager's acid-growth hypothesis. Despite the accumulation of observations that evidently identify the final target of the auxin-induced action to be H +-ATPase, which excretes H + protons to the apoplast and take in K + ions through its rectifying K + channel in the following years, the ...