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The brainstem (or brain stem) is the posterior stalk-like part of the brain that connects the cerebrum with the spinal cord. [1] In the human brain the brainstem is composed of the midbrain , the pons , and the medulla oblongata . [ 1 ]
Central pattern generators (CPGs) are self-organizing biological neural circuits [1] [2] that produce rhythmic outputs in the absence of rhythmic input. [3] [4] [5] They are the source of the tightly-coupled patterns of neural activity that drive rhythmic and stereotyped motor behaviors like walking, swimming, breathing, or chewing.
Sectional organization of spinal cord. The spinal cord is the main pathway for information connecting the brain and peripheral nervous system. [3] [4] Much shorter than its protecting spinal column, the human spinal cord originates in the brainstem, passes through the foramen magnum, and continues through to the conus medullaris near the second lumbar vertebra before terminating in a fibrous ...
reticulospinal tract: connects the reticular system, a diffuse region of gray matter in the brain stem, to the spinal cord. It also contributes to muscle tone and influences autonomic functions. lateral vestibulospinal tract: Connects the brain stem nuclei of the vestibular system with the spinal cord. This allows posture, movement, and balance ...
[11] [12] Of particular note are implanted electrical devices such as cardiac pacemakers or defibrillators or other implanted stimulators such as deep brain stimulators or spinal cord stimulators. [ 11 ] [ 12 ] Theoretically, delivering electricity through the body may affect systems in the body that depend on electrical signals, such as the ...
Ataxia (from Greek α- [a negative prefix] + -τάξις [order] = "lack of order") is a neurological sign consisting of lack of voluntary coordination of muscle movements that can include gait abnormality, speech changes, and abnormalities in eye movements, that indicates dysfunction of parts of the nervous system that coordinate movement, such as the cerebellum.
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This mutant protein causes degradation in certain types of neurons, like Purkinje neurons, which are common in the cerebellum, spinal cord, and related parts of the brain. While the mechanism is not fully understood, it is suspected that changes in the interactions between ataxin 1 and other proteins result in a toxic gain of function.