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  2. Ketonuria - Wikipedia

    en.wikipedia.org/wiki/Ketonuria

    Any diabetic patient who has elevated levels of blood and urine glucose should be tested for urinary ketones. In addition, when diabetic treatment is being switched from insulin to oral hypoglycemic agents, the patient's urine should be monitored for ketonuria. The development of ketonuria within 24 hours after insulin withdrawal usually ...

  3. Diabetic ketoacidosis - Wikipedia

    en.wikipedia.org/wiki/Diabetic_ketoacidosis

    In general, insulin is given at 0.1 units/kg per hour to reduce blood sugars and suppress ketone production. Guidelines differ as to which dose to use when blood sugar levels start falling; American guidelines recommend reducing the dose of insulin once glucose falls below 16.6 mmol/L (300 mg/dL) [ 3 ] and UK guidelines at 14 mmol/L (253 mg/dL ...

  4. Ketosis - Wikipedia

    en.wikipedia.org/wiki/Ketosis

    Ketosis is a metabolic state characterized by elevated levels of ketone bodies in the blood or urine. Physiological ketosis is a normal response to low glucose availability. . In physiological ketosis, ketones in the blood are elevated above baseline levels, but the body's acid–base homeostasis is maintain

  5. Insulin Resistance: From Symptoms to Treatment - AOL

    www.aol.com/insulin-resistance-symptoms...

    This one measures blood sugar after fasting and again two hours after you drink a sugary drink. The fasting plasma glucose and oral glucose tolerance tests are more sensitive than the A1C test.

  6. Hyperinsulinemic hypoglycemia - Wikipedia

    en.wikipedia.org/wiki/Hyperinsulinemic_hypoglycemia

    Oral administration of glucose can worsen the outcome, as more insulin is eventually produced. Most people recover fully even from severe hypoglycemia after the blood glucose is restored to normal. Recovery time varies from minutes to hours depending on the severity and duration of the hypoglycemia.

  7. Complications of diabetes - Wikipedia

    en.wikipedia.org/wiki/Complications_of_diabetes

    In the early days of insulin treatment for type 1 diabetes there was much debate as to whether strict control of hyperglycaemia would delay or prevent the long-term complications of diabetes. The work of Pirart [ 50 ] suggested that microvascular complications of diabetes were less likely to occur in individuals with better glycaemic control.

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