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  2. Hyperkalemia - Wikipedia

    en.wikipedia.org/wiki/Hyperkalemia

    A standard ampule of 10% calcium gluconate is also 10 mL but has only 2.26 mmol of calcium. Clinical practice guidelines recommend giving 6.8 mmol for typical EKG findings of hyperkalemia. [13] This is 10 mL of 10% calcium chloride or 30 mL of 10% calcium gluconate. [13]

  3. Hyperkalemic periodic paralysis - Wikipedia

    en.wikipedia.org/wiki/Hyperkalemic_periodic...

    Hyperkalemic periodic paralysis causes episodes of extreme muscle weakness, with attacks often beginning in childhood. [1] Depending on the type and severity of the HyperKPP, it can increase or stabilize until the fourth or fifth decade where attacks may cease, decline, or, depending on the type, continue on into old age.

  4. Pseudohypoaldosteronism - Wikipedia

    en.wikipedia.org/wiki/Pseudohypoaldosteronism

    Renal salt wasting and high concentrations of sodium in sweat, stool, and saliva. The disorder often involves multiple organ systems and can be life threatening in the neonatal period. Patients usually present with hyponatremia, hyperkalemia, and increased plasma renin activity with high serum aldosterone concentrations.

  5. Electrocardiography - Wikipedia

    en.wikipedia.org/wiki/Electrocardiography

    ICD-10-PCS: R94.31: ICD-9-CM: 89.52: MeSH: ... Conversely, a discovery of hyperkalemia should be followed by an ECG for manifestations such as peaked T waves, widened ...

  6. Hypoaldosteronism - Wikipedia

    en.wikipedia.org/wiki/Hypoaldosteronism

    Hypoaldosteronism causes low sodium (hyponatremia), high potassium (hyperkalemia), and metabolic acidosis, a condition in which the body produces excess acid.These conditions are responsible for the symptoms of hypoaldosteronism, which include muscle weakness, nausea, palpitations, irregular heartbeat, and abnormal blood pressure.

  7. Hyperaldosteronism - Wikipedia

    en.wikipedia.org/wiki/Hyperaldosteronism

    Secondary hyperaldosteronism (also hyperreninism, or hyperreninemic hyperaldosteronism) is due to overactivity of the renin–angiotensin–aldosterone system (RAAS).. The causes of secondary hyperaldosteronism are accessory renal veins, fibromuscular dysplasia, reninoma, renal tubular acidosis, nutcracker syndrome, ectopic tumors, massive ascites, left ventricular failure, and cor pulmonale.

  8. Periodic paralysis - Wikipedia

    en.wikipedia.org/wiki/Periodic_paralysis

    Periodic paralysis is an autosomal dominant myopathy with considerable variation in penetrance, leading to a spectrum of familial phenotypes (only one parent needs to carry the gene mutation to affect the children, but not all family members who share the gene are affected to the same degree).

  9. Electrolyte imbalance - Wikipedia

    en.wikipedia.org/wiki/Electrolyte_imbalance

    [9] [10] [11] Kidneys work to keep the electrolyte concentrations in blood constant despite changes in the body. [6] [8] For example, during heavy exercise, electrolytes are lost in sweat, particularly in the form of sodium and potassium. [8] The kidneys can also generate dilute urine to balance sodium levels. [8]