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Peribronchial cuffing, also referred to as peribronchial thickening or bronchial wall thickening, is a radiologic sign which occurs when excess fluid or mucus buildup in the small airway passages of the lung causes localized patches of atelectasis (lung collapse). [1] This causes the area around the bronchus to appear more prominent on an X-ray ...
A chest x-ray is abnormal in most patients with bronchiectasis. Computed tomography is recommended to confirm the diagnosis and is also used to describe the distribution and grade the severity of the disease. Radiographic findings include airway dilation, bronchial wall thickening, and atelectasis. [65]
The findings on chest imaging in DIPNECH patients are bilateral and diffuse. The most frequent findings on a computed tomography (CT) of the chest are multiple primary nodules and/or masses, on a background of mosaic attenuation and airway wall thickening. [2] [9] The nodules have an indolent pattern of growth and are found throughout the lungs.
Tram tracks are caused by bronchial wall thickening, and can be detected on a lateral chest X-ray. [1] Nephrology
The double bronchial wall sign is best visualized on CT, which provides high-resolution images of the mediastinal structures. The double bronchial wall sign is commonly seen in the central bronchi, particularly in the trachea or mainstem bronchi, where air is more likely to outline the structures.
Pleural thickening - Irregularity or abnormal prominence of the pleural margin, including apical capping (thickening of the pleura in the apical region). Pleural thickening can be calcified. Diaphragmatic tenting - A localized accentuation of the normal convexity of the hemidiaphragm as if "pulled upwards by a string."
Bronchial wall thickness (T) and bronchial diameter (D). Bronchial wall thickening, as can be seen on CT scan, generally (but not always) implies inflammation of the bronchi . [15] Normally, the ratio of the bronchial wall thickness and the bronchial diameter is between 0.17 and 0.23. [16]
Obstruction of the lumen of the bronchiole by mucoid exudate, goblet cell metaplasia, epithelial basement membrane thickening and severe inflammation of bronchiole. During an asthma episode, inflamed airways react to environmental triggers such as smoke, dust, or pollen. The airways narrow and produce excess mucus, making it difficult to breathe.