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Toxic encephalopathy is a neurologic disorder caused by exposure to neurotoxic organic solvents such as toluene, following exposure to heavy metals such as manganese, as a side effect of melarsoprol treatment for African trypanosomiasis, adverse effects to prescription drugs, or exposure to extreme concentrations of any natural toxin such as cyanotoxins found in shellfish or freshwater ...
A metabolic encephalopathy develops, and this can progress to coma and death without treatment. [4] Ammonia is only toxic to the brain, other tissues can handle elevated ammonia concentrations without problems. [5] Later onset forms of OTC deficiency can have variable presentations.
Toxic-metabolic encephalopathy: A catch-all for brain dysfunction caused by infection, organ failure, or intoxication. Transmissible spongiform encephalopathy : A collection of diseases all caused by prions, and characterized by "spongy" brain tissue (riddled with holes), impaired locomotion or coordination, and a 100% mortality rate.
Chronic solvent-induced encephalopathy (CSE) is a condition induced by long-term exposure to organic solvents, often—but not always—in the workplace, that lead to a wide variety of persisting sensorimotor polyneuropathies and neurobehavioral deficits even after solvent exposure has been removed.
From November 1995 to November 1996 in France, a national survey of pediatric departments for children under 15 years of age with unexplained encephalopathy and a threefold (or greater) increase in serum aminotransferase and/or ammonia led to the identification of nine definite cases of Reye syndrome (0.79 cases per million children). Eight of ...
Cerebral edema is present with many common cerebral pathologies and risk factors for development of cerebral edema will depend on the cause. [1] The following were reliable predictors for development of early cerebral edema in ischemic strokes. [9] [10] Younger age; Higher severity of symptoms on the National Institutes of Health Stroke Scale
The Commission’s new report added high cholesterol after the age of 40 and vision loss as new risk factors to that list, suggesting that they contribute to about 9% of all dementia cases — 7% ...
Patients presenting as acute and hyperacute liver failure are at greater risk of developing cerebral edema and grade IV encephalopathy. The pathogenesis remains unclear, but is likely to be a consequence of several phenomena. There is a buildup of toxic substances like ammonia, mercaptan, serotonin and tryptophan in the brain.