Search results
Results From The WOW.Com Content Network
Bronze disease is an irreversible and nearly inexorable corrosion process that occurs when chlorides come into contact with bronze or other copper-bearing alloys. [1] It can occur as both a dark green coating, or as a much lighter whitish fuzzy or furry green coating. [ 1 ]
BTA is used in heritage conservation, notably for the treatment of bronze disease. The exact structure of the copper-BTA complex is controversial and many proposals have been suggested. [citation needed] Chemical structure of the coordination polymer from benzotriazolate and copper(I), the active ingredient in the BT-derived corrosion inhibition
Bronze artifacts from Sumerian cities and Egyptian artifacts of copper and bronze alloys date to 3000 BC. [10] The Bronze Age began in Southeastern Europe around 3700–3300 BC, in Northwestern Europe about 2500 BC. It ended with the beginning of the Iron Age, 2000–1000 BC in the Near East, 600 BC in Northern Europe.
The Statue of Liberty, showing advanced patination; verdigris is responsible for the statue's iconic green colour.. Verdigris (/ ˈ v ɜːr d ɪ ɡ r iː (s)/) [1] is a common name for any of a variety of somewhat toxic [2] [3] [4] copper salts of acetic acid, which range in colour from green to a bluish-green depending on their chemical composition.
The conservation and restoration of outdoor bronze artworks is an activity dedicated to the preservation, protection, and maintenance of bronze objects and artworks that are on view outside. When applied to cultural heritage this activity is generally undertaken by a conservator-restorer .
Bronze typically oxidizes only superficially; once a copper oxide (eventually becoming copper carbonate) layer is formed, the underlying metal is protected from further corrosion. This can be seen on statues from the Hellenistic period. If copper chlorides are formed, a corrosion-mode called "bronze disease" will eventually destroy it ...
A 1997 review (Suarez-Almazor ME, et al) [6] reports that treatment with intramuscular gold (parenteral gold) reduces disease activity and joint inflammation. Gold-containing drugs taken by mouth are less effective than by injection. Three to six months are often required before gold treatment noticeably improves symptoms.
One of the difficulties with ALD as a disease included in universal newborn screening is the difficulty in predicting the eventual phenotype that an individual will express. The accepted treatment for affected boys presenting with the cerebral childhood form of the disease is a bone marrow transplant, a procedure which carries significant risks.