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Stunted growth, also known as stunting or linear growth failure, is defined as impaired growth and development manifested by low height-for-age. [1] It is a manifestation of malnutrition (undernutrition) and can be caused by endogenous factors (such as chronic food insecurity) or exogenous factors (such as parasitic infection ).
Renal agenesis is a medical condition in which one (unilateral) or both (bilateral) fetal kidneys fail to develop. Unilateral and bilateral renal agenesis in humans, mice and zebra fish has been linked to mutations in the gene GREB1L. [1] It has also been associated with mutations in the genes RET or UPK3A [2] in humans [3] and mice respectively.
The kidneys can be unilobar (a single lobe represented by a single renal pyramid) or multilobar, [13] [14] unipapillary (a single or a common papilla), with several papillae or multipapillary, [14] [15] may be smooth-surfaced or lobulated. [1] [13] The multilobar kidneys can also be reniculate, which are found mainly in marine mammals. [16]
Unlike chronic kidney disease, however, the kidneys can often recover from acute kidney injury, allowing the person with AKI to resume a normal life. People with acute kidney injury require supportive treatment until their kidneys recover function, and they often remain at increased risk of developing future kidney failure.
The development of the kidney proceeds through a series of successive phases, each marked by the development of a more advanced kidney: the archinephros, pronephros, mesonephros, and metanephros. [1] The pronephros is the most immature form of kidney, while the metanephros is most developed. The metanephros persists as the definitive adult kidney.
The vas deferens can grow back together after a vasectomy–thus resulting in vasectomy failure. [40] This occurs due to the fact that the epithelium of the vas deferens, similar to the epithelium of some other human body parts, is capable of regenerating and creating a new tube in the event that the vas deferens is damaged and/or severed. [41]