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For example, in the context of depression, the diathesis-stress model can help explain why Person A may become depressed while Person B does not, even when exposed to the same stressors. [7] More recently, the diathesis-stress model has been used to explain why some individuals are more at risk for developing a disorder than others. [9]
For example, those carrying a larger number of sensitivity genes may develop a sensitivity that is more geared towards threat when growing up in a challenging or adverse environment (i.e., vulnerability as described in the Diathesis-Stress model), whereas those growing up in a predominately supportive and secure context may develop heightened ...
The idea that individuals vary in their sensitivity to their environment was historically framed in diathesis-stress [4] or dual-risk terms. [5] These theories suggested that some "vulnerable" individuals, due to their biological, temperamental and/or physiological characteristics (i.e., "diathesis" or "risk 1"), are more vulnerable to the adverse effects of negative experiences (i.e., "stress ...
The diathesis-stress model is an interactionism approach. In the context of schizophrenia, diathesis is the vulnerability. Vulnerabilities can be either genetic predisposition or early experiences, or both. [22] Stressors are any event that can trigger a schizophrenia-vulnerable individual to the onset of the condition.
A related view, the diathesis-stress model, posits that mental disorders result from genetic dispositions and environmental stressors, combining to cause patterns of distress or dysfunction. [19] The model is one way to explain why some individuals are more vulnerable to mental disorders than others.
The diathesis–stress model [23] emphasizes the importance of applying multiple causality to psychopathology, by stressing that disorders are caused by both precipitating causes, and predisposing causes. A precipitating cause is an immediate trigger that instigates a person's action or behavior.
Stress can contribute to an increased appetite, food cravings, and weight gain, especially around the stomach. It can also mess with sleep, zap motivation to work out, and trigger stress eating .
[5] [37] The diathesis–stress model specifies that depression results when a preexisting vulnerability, or diathesis, is activated by stressful life events. The preexisting vulnerability can be either genetic , [ 38 ] [ 39 ] implying an interaction between nature and nurture , or schematic , resulting from views of the world learned in ...