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The diathesis-stress model, also known as the vulnerability–stress model, is a psychological theory that attempts to explain a disorder, or its trajectory, as the result of an interaction between a predispositional vulnerability, the diathesis, and stress caused by life experiences.
The concept of Environmental Sensitivity integrates multiple theories on how people respond to negative and positive experiences. These include the frameworks of Diathesis-stress model [4] and Vantage Sensitivity, [5] as well as the three leading theories on more general sensitivity: Differential Susceptibility, [6] [7] Biological Sensitivity to Context, [8] and Sensory processing sensitivity ...
The differential susceptibility theory proposed by Jay Belsky [1] is another interpretation of psychological findings that are usually discussed according to the diathesis-stress model. Both models suggest that people's development and emotional affect are differentially affected by experiences or qualities of the environment. Where the ...
The diathesis-stress model is an interactionism approach. In the context of schizophrenia, diathesis is the vulnerability. Vulnerabilities can be either genetic predisposition or early experiences, or both. [22] Stressors are any event that can trigger a schizophrenia-vulnerable individual to the onset of the condition.
A related view, the diathesis-stress model, posits that mental disorders result from genetic dispositions and environmental stressors, combining to cause patterns of distress or dysfunction. [19] The model is one way to explain why some individuals are more vulnerable to mental disorders than others.
The diathesis–stress model [23] emphasizes the importance of applying multiple causality to psychopathology, by stressing that disorders are caused by both precipitating causes, and predisposing causes. A precipitating cause is an immediate trigger that instigates a person's action or behavior.
[5] [37] The diathesis–stress model specifies that depression results when a preexisting vulnerability, or diathesis, is activated by stressful life events. The preexisting vulnerability can be either genetic , [ 38 ] [ 39 ] implying an interaction between nature and nurture , or schematic , resulting from views of the world learned in ...
The investigators suggested that in utero insults may specifically stress those with a schizophrenia diathesis, supporting a "diathesis-stress" etiological model for a subset of schizophrenia (that Kraepelin identified) with early abnormalities suggesting brain atrophy. [57]