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Vipera berus - Venom delivery apparatus. Snake venom is a highly toxic saliva [1] containing zootoxins that facilitates in the immobilization and digestion of prey. This also provides defense against threats. Snake venom is usually injected by unique fangs during a bite, though some species are also able to spit venom. [2]
Side effects may be severe. [1] They include serum sickness, shortness of breath, and allergic reactions including anaphylaxis. [1] Antivenom is traditionally made by collecting venom from the relevant animal and injecting small amounts of it into a domestic animal. [2]
Anti-aging creams are predominantly moisturizer-based skin care products marketed with unproven claims of making the consumer look younger by reducing, masking or preventing signs of skin aging. Anti-aging supplements are ingestible products promoted to diminish the effects of aging, including vitamin supplements , powders, and teas.
The venom of L. acrochorda is extremely dangerous in that a snakebite incident will result in a 90% chance of death. Its venom can cause serious side effects, such as vasodilation and blood anti-coagulation. [4]
Crotoxin (CTX) is the main toxic compound in the snake venom of the South American rattlesnake, Crotalus durissus terrificus.Crotoxin is a heterodimeric beta-neurotoxin, composed of an acidic, non-toxic and non-enzymatic subunit (CA), and a basic, weakly toxic, phospholipase A2 protein (CB).
Venom-induced consumption coagulopathy (VICC) is a medical condition caused by the effects of some snake and caterpillar venoms on the blood. [1] Important coagulation factors are activated by the specific serine proteases in the venom and as they become exhausted, coagulopathy develops. Symptoms are consistent with uncontrolled bleeding.
Taipoxin is a potent myo- and neurotoxin that was isolated from the venom of the coastal taipan Oxyuranus scutellatus or also known as the common taipan. [1] Taipoxin like many other pre-synaptic neurotoxins are phospholipase A 2 (PLA 2) toxins, which inhibit/complete block the release of the motor transmitter acetylcholine and lead to death by paralysis of the respiratory muscles (). [2]