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The causes of acute kidney injury are commonly categorized into prerenal, intrinsic, and postrenal. Acute kidney injury occurs in up to 30% of patients following cardiac surgery. [ 22 ] Mortality increases by 60-80% in post-cardiopulmonary bypass patients who go on to require renal replacement therapy.
This can again cause blood to be in the urine, or hematuria, and flank pain. Other potential causes of renal papillary necrosis are chronic use of analgesics, like aspirin, as well as diabetes mellitus, sickle cell disease, and pyelonephritis. In general, with intrarenal AKI, the kidneys lose the ability to filter the blood properly.
Acute tubular necrosis (ATN) is a medical condition involving the death of tubular epithelial cells that form the renal tubules of the kidneys.Because necrosis is often not present, the term acute tubular injury (ATI) is preferred by pathologists over the older name acute tubular necrosis (ATN). [1]
Renal azotemia (acute kidney failure) typically leads to uremia. It is an intrinsic disease of the kidney, generally the result of kidney parenchymal damage. Causes include kidney failure, glomerulonephritis, acute tubular necrosis, or other kidney disease. [3] The BUN:Cr in renal azotemia is less than 15.
Kidney ischemia [1] is a disease with a high morbidity and mortality rate. [2] Blood vessels shrink and undergo apoptosis which results in poor blood flow in the kidneys. More complications happen when failure of the kidney functions result in toxicity in various parts of the body which may cause septic shock, hypovolemia, and a need for surgery. [3]
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