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Silent sinus syndrome is a subtype of stage three chronic maxillary atelectasis. The distinguishing factor is that in silent sinus syndrome, there is an absence of sinusitis symptoms. [3] [4] [5] To be clear, chronic maxillary sinusitis may be a primary causitive factor in a significant number of silent sinus syndrome cases, it just may be ...
Special views focusing on the orbit of the eye may be taken to investigate concerns relating to the eye. [8] CT scans are used by physicians specializing in treating the eye (ophthalmologists) to detect foreign bodies (especially metallic objects), fractures, abscesses, cellulitis, sinusitis, bleeding within the skull (intracranial bleeding), proptosis, Graves disease changes in the eye, and ...
CT scan can show the full extent of the polyp, which may not be fully appreciated with physical examination alone. Imaging is also required for planning surgical treatment. [7] On a CT scan, a nasal polyp generally has an attenuation of 10–18 Hounsfield units, which is similar to that of mucus. Nasal polyps may have calcification. [14]
Maxillary sinuses. Frontal sinuses, seen with an oblique view. Ethmoidal cells. Sphenoid sinus, seen through the open mouth. Odontoid process, where if it is just below the mentum, it confirms adequate extension of the head. The frontal sinus may not show the frontal sinus in detail. [1]
For sinusitis lasting more than 12 weeks, a CT scan is recommended. [65] On a CT scan, acute sinus secretions have a radiodensity of 10 to 25 Hounsfield units (HU), but in a more chronic state they become more viscous, with a radiodensity of 30 to 60 HU. [67] Nasal endoscopy and clinical symptoms are also used to make a positive diagnosis. [28]
The maxillary sinuses, the largest of the paranasal sinuses, are under the eyes, in the maxillary bones (open in the back of the semilunar hiatus of the nose). They are innervated by the maxillary nerve (CN V2). [2] The frontal sinuses, superior to the eyes, in the frontal bone, which forms the hard part of the forehead.
The typical symptoms of outward expansion are prominence of the supraorbital ridge and frontal bossing. The expansion could be focused on the orbit, nose, and other sinuses, or it could go intracranially. Sinus pressure, diplopia, ocular abnormalities, anosmia, and headache are associated symptoms. [1]
Microcornea, glaucoma, and absent frontal sinuses is a very rare developmental genetic disorder that occurs during embryogenesis which is characterized by a combination of microcornea, glaucoma and missing/underdeveloped sinuses. Additional findings include increased palmar skin thickness and torus palatinus.
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