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Betel nut chewing, also called betel quid chewing or areca nut chewing, is a practice in which areca nuts (also called "betel nuts") are chewed together with slaked lime and betel leaves for their stimulant and narcotic effects, the primary psychoactive compound being arecoline.
In order to chew maak the traditional Thai way, three main ingredients are needed: betel leaf, betel nut and red limestone paste. [4] Before a betel chew, the betel nut is boiled, sliced and dried. [1] A popular method is to cut the betel nut into four smaller sections before solar drying, since betel nut can be very strong.
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The practice of betel nut chewing, often together with other herbs as a stimulant drug, dates back thousands of years, and continues to the present day in many countries. Betel nut chewing is addictive due to the presence of the stimulant arecoline, and causes adverse health effects, mainly oral and esophageal cancers, and
Betel nuts were introduced into Hunan, the biggest consumer of betel nuts, more than 300 years ago and embedded in the social culture of the region that they were listed as a provincial-level cultural heritage in 2016. [4] The Opium Wars and the Taiping Rebellion destabilized the national betel nut trade. Betel nut chewing would not experience ...
English: Paan, the betel leaf and areca nut preparation for chewing, causes notable psychoactive effects. This preparation widely consumed throughout South Asia and Southeast Asia. It is chewed for its stimulant and psychoactive effects.
Betel leaves are also used as to wrap betel quid for chewing, which also contains the toxic and mildly narcotic areca nut. [16] Habitual use of this popular product (sometimes inaccurately referred to as "betel nut") damages the oral cavity and is associated with a wide range of adverse systemic health effects, including harm to the ...
Arecoline is the primary active ingredient responsible for the central nervous system effects of the areca nut. Arecoline has been compared to nicotine; however, nicotine agonizes nicotinic acetylcholine receptors, whereas arecoline is primarily a partial agonist of muscarinic acetylcholine receptors, [4] [5] leading to its parasympathetic effects.