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2. Stroke recovery - Wikipedia

   en.wikipedia.org/wiki/Stroke_recovery

   [48] [51] [52] MSC treatment also appears to improve the control of cerebral blood flow and blood–brain barrier permeability, [53] [54] as well as what is currently thought to be the most important mechanism of MSC treatment after stroke, the activation of endogenous neuroprotection and neurorestoration pathways by the release of cytokines ...

3. Dejerine–Roussy syndrome - Wikipedia

   en.wikipedia.org/wiki/Dejerine–Roussy_syndrome

   Treatments last 4–12 hours and in general are well tolerated, and the occurrence of adverse events does not differ significantly across patients. Commonly reported side-effects are dizziness, decreased intellectual performance, somnolence, and nausea. [1] Topical treatment such as lidocaine patches can be used to treat pain locally.

4. Your biggest questions about strokes, answered - AOL

   www.aol.com/biggest-questions-strokes-answered...

   The most common treatment for ischemic stroke is injecting a medicine into a vein in your arm that breaks up blood clots. You must get to hospital within the first three hours of noticing symptoms ...

5. Sensory stimulation therapy - Wikipedia

   en.wikipedia.org/wiki/Sensory_stimulation_therapy

   Sensory stimulation therapy (SST) is an experimental therapy that aims to use neural plasticity mechanisms to aid in the recovery of somatosensory function after stroke or cognitive ageing. Stroke and cognitive ageing are well known sources of cognitive loss, the former by neuronal death, the latter by weakening of neural connections. SST ...

6. Tenecteplase more likely to lead to 'excellent' stroke ...

   www.aol.com/tenecteplase-more-likely-lead...

   A recent paper reports that the use of the drug tenecteplase, not yet approved for stroke treatment in the U.S., may lead to slightly better outcomes after a stroke than the commonly used alteplase.

7. Cerebral edema - Wikipedia

   en.wikipedia.org/wiki/Cerebral_edema

   The current understanding of the pathophysiology of cerebral edema after traumatic brain injury or intracerebral hemorrhage is incomplete. [8] [54] Current treatment therapies aimed at cerebral edema and increased intracranial pressure are effective at reducing intracranial hypertension but have unclear impacts on functional outcomes. [53]