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  2. Acetylcysteine - Wikipedia

    en.wikipedia.org/wiki/Acetylcysteine

    Acetylcysteine is extensively liver metabolized, CYP450 minimal, urine excretion is 22–30% with a half-life of 5.6 hours in adults and 11 hours in newborns. [medical citation needed] Acetylcysteine is the N-acetyl derivative of the amino acid L-cysteine, and is a precursor in the formation of the antioxidant glutathione in the body.

  3. Rumack–Matthew nomogram - Wikipedia

    en.wikipedia.org/wiki/Rumack–Matthew_nomogram

    When the NAC study began in 1976, the U.S. Food and Drug Administration (FDA) required a line that was 25% below the original. This line is referred to as the treatment line and starts at 150 μg/mL at 4 hours. It is the usual line used in the United States to determine treatment of acetaminophen overdose after the publication of the NAC study ...

  4. NAPQI - Wikipedia

    en.wikipedia.org/wiki/NAPQI

    In adults, the primary metabolic pathway for paracetamol is glucuronidation. [1] This yields a relatively non-toxic metabolite, which is excreted into bile and passed out of the body. A small amount of the drug is metabolized via the cytochrome P-450 pathway (to be specific, CYP3A4 and CYP2E1 ) into NAPQI, which is extremely toxic to liver ...

  5. Pharmacokinetics - Wikipedia

    en.wikipedia.org/wiki/Pharmacokinetics

    The rate at which a drug enters into the body for oral and other extravascular routes. ⁡ 0.693 h −1: Elimination half-‍life: The time required for the concentration of the drug to reach half of its original value.

  6. N-acetyl-cysteine - Wikipedia

    en.wikipedia.org/?title=N-acetyl-cysteine&...

    This page was last edited on 21 June 2011, at 21:08 (UTC).; Text is available under the Creative Commons Attribution-ShareAlike 4.0 License; additional terms may ...

  7. Cystine/glutamate transporter - Wikipedia

    en.wikipedia.org/wiki/Cystine/glutamate_transporter

    N-acetylcysteine has been shown to decrease drug-seeking behavior for nicotine and heroin as well. However, N-acetylcysteine does not alter the cocaine-induced rush or euphoria; it only causes a reduction in drug-seeking behavior. N-acetylcysteine works by increasing levels of cysteine in cells, leading to an increase in system Xc- activity.