Ad
related to: how long is hpv dormant
Search results
Results From The WOW.Com Content Network
Some HPV types, such as HPV5, may establish infections that persist for the lifetime of the individual without ever manifesting any clinical symptoms. HPV types 1 and 2 can cause common warts in some infected individuals. [18] HPV types 6 and 11 can cause genital warts and laryngeal papillomatosis. [1] Many HPV types are carcinogenic. [19]
“HPV can be dormant and not detected for many years,” says Dr. Sarah H. Kim, division director of gynecologic oncology at Penn Medicine. “Then, for whatever reason, it can reactivate and ...
Why do some people develop cancer from HPV but others do not? Martina Navratilova has throat caused by the human papillomavirus (HPV). ... and although it may remain dormant for a really long time ...
Virus latency (or viral latency) is the ability of a pathogenic virus to lie dormant within a cell, denoted as the lysogenic part of the viral life cycle. [1] A latent viral infection is a type of persistent viral infection which is distinguished from a chronic viral infection. Latency is the phase in certain viruses' life cycles in which ...
Papillomaviridae is a family of non-enveloped DNA viruses whose members are known as papillomaviruses. [1] Several hundred species of papillomaviruses, traditionally referred to as "types", [2] have been identified infecting all carefully inspected mammals, [2] but also other vertebrates such as birds, snakes, turtles and fish.
For premium support please call: 800-290-4726 more ways to reach us
[13] [14] However, it is uncertain whether oral sex would directly transmit the virus [14] or activate the dormant virus that was transmitted at childbirth. [14] [13] In general, physicians are unsure why only certain people who have been exposed to the HPV types implicated in the disease develop laryngeal papillomatosis.
Human HPV has long been implicated in the pathogenesis of several anogenital cancers including those of the anus, vulva, vagina, cervix, and penis. [13] In 2007 it was also implicated by both molecular and epidemiological evidence in cancers arising outside of the anogenital tract, namely oral cancers.