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In some cases stroke and transient ischemic attacks have occurred after sclerotherapy. [17] Varicose veins and reticular veins are often treated before treating telangiectasia, although treatment of these larger veins in advance of sclerotherapy for telangiectasia may not guarantee better results.
Treatment treatment depends on cause An esophageal motility disorder ( EMD ) is any medical disorder resulting from dysfunction of the coordinated movement of esophagus, which causes dysphagia (i.e. difficulty in swallowing , regurgitation of food).
A treatment used sometimes is endoscopic band ligation. [27] In 2010, a team of Japanese surgeons performed a "novel endoscopic ablation of gastric antral vascular ectasia". [10] The experimental procedure resulted in "no complications". [10] Relapse is possible, even after treatment by argon plasma coagulation and progesterone. [21]
Treatment Endoscopy with argon plasma coagulation , bipolar electrocautery, radiofrequency ablation Radiation proctitis or radiation proctopathy is a condition characterized by damage to the rectum after exposure to x-rays or other ionizing radiation as a part of radiation therapy . [ 1 ]
Generalized essential telangiectasia, also known as general essential telangiectasia, [1] is characterized by the dilation of veins and capillaries over a large segment of the body without preceding or coexisting lesions, telengiectases that may be distributed over the entire body or be localized to some large area such as the legs, arms, or trunk.
Lesions lips, patient with hemorrhagic hereditary telangiectasia. Hereditary hemorrhagic telangiectasia (HHT), also known as Osler–Weber–Rendu disease and Osler–Weber–Rendu syndrome, is a rare autosomal dominant genetic disorder that leads to abnormal blood vessel formation in the skin, mucous membranes, and often in organs such as the lungs, liver, and brain.
Unilateral nevoid telangiectasia can be congenital or acquired. Rare congenital type manifests at or soon after the neonatal period; it is more common in males and occurs in an autosomal dominant pattern. Conversely, the acquired form is nearly exclusively found in young female patients who have physiologic problems. [7]
Telangiectasia macularis eruptiva perstans (TMEP) is persistent, pigmented, asymptomatic eruption of macules usually less than 0.5 cm in diameter with a slightly reddish-brown tinge. [ 1 ] : 616 [ 2 ]