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This is an accepted version of this page This is the latest accepted revision, reviewed on 7 January 2025. Circumstances, mechanisms, and factors of tobacco consumption on human health "Health effects of smoking" and "Dangers of smoking" redirect here. For cannabis, see Effects of cannabis. For smoking crack cocaine, see Crack cocaine § Health issues. "Smoking and health" redirects here. For ...
Smoking is a practice in which a substance is combusted and the resulting smoke is typically inhaled to be tasted and absorbed into the bloodstream of a person. Most commonly, the substance used is the dried leaves of the tobacco plant, which have been rolled with a small rectangle of paper into an elongated cylinder called a cigarette.
Tobacco smoking is the leading cause of preventable death and a global public health concern. [86] There are 1.3 billion tobacco users in the world, as per latest data from WHO. [17] One person dies every six seconds from a tobacco related disease. [87] Common adverse effects of tobacco smoking. The more common effects are in bold face. [88]
The consumption of tobacco products and its harmful effects affect both smokers and non-smokers, [9] and is a major risk factor for six of the eight leading causes of deaths in the world, including respiratory diseases, cardiovascular diseases, cerebrovascular diseases, periodontal diseases, teeth decay and loss, over 20 different types or subtypes of cancers, strokes, several debilitating ...
In 2003, in response to growth of tobacco use in developing countries, the World Health Organization [26] successfully rallied 168 countries to sign the Framework Convention on Tobacco Control. The convention is designed to push for effective legislation and enforcement in all countries to reduce the harmful effects of tobacco. [27]
The effects on sleep vary after being intoxicated, during withdrawal, and from long-term use. [26] Nicotine may result in arousal and wakefulness, mainly via incitement in the basal forebrain. [27] Nicotine withdrawal, after abstaining from nicotine use in non-smokers, was linked with longer overall length of sleep and REM rebound. [26]
One of the most prominent and well studied epigenetic consequences of cigarette smoke is altered DNA methylation. Cigarette smoke acts through a number of mechanisms to effect this, chief among these being smoke-induced damage to the DNA and altered expression levels of proteins involved in DNA methylation and transcriptional regulation.
A National Institute on Drug Abuse video entitled Anyone Can Become Addicted to Drugs. [21]Nicotine dependence is defined as a neurobiological adaptation to repeated drug exposure that is manifested by highly controlled or compulsive use, the development of tolerance, experiencing withdrawal symptoms upon cessation including cravings, and an inability to quit despite harmful effects. [9]