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  2. Equianalgesic - Wikipedia

    en.wikipedia.org/wiki/Equianalgesic

    An equianalgesic chart is a conversion chart that lists equivalent doses of analgesics (drugs used to relieve pain). Equianalgesic charts are used for calculation of an equivalent dose (a dose which would offer an equal amount of analgesia) between different analgesics. [1]

  3. Antithyroid agent - Wikipedia

    en.wikipedia.org/wiki/Antithyroid_agent

    They inhibit release of thyroid hormones by the thyroid gland.The most studied drug in this class is lithium, which inhibits thyroid hormone secretion by inhibiting iodotyrosine coupling, thyroidal iodide uptake, and alteration in structure of thyroglobulin, [10] a protein which acts as a substrate for the synthesis of thyroid hormones and storage of inactive forms of T3, T4 and iodine within ...

  4. ATC code H03 - Wikipedia

    en.wikipedia.org/wiki/ATC_code_H03

    ATC code H03 Thyroid therapy is a therapeutic subgroup of the Anatomical Therapeutic Chemical Classification System, a system of alphanumeric codes developed by the World Health Organization (WHO) for the classification of drugs and other medical products.

  5. Liothyronine - Wikipedia

    en.wikipedia.org/wiki/Liothyronine

    Liothyronine may be used when there is an impaired conversion of T 4 to T 3 in peripheral tissues. [2] The dose of liothyronine for hypothyroidism is a lower amount than levothyroxine due it being a higher concentrated synthetic medication. [2] About 25 μg of liothyronine is equivalent to 100 μg of levothyroxine. [3]

  6. ATC code H - Wikipedia

    en.wikipedia.org/wiki/ATC_code_H

    ATC code H Systemic hormonal preparations, excluding sex hormones and insulins is a section of the Anatomical Therapeutic Chemical Classification System, a system of alphanumeric codes developed by the World Health Organization (WHO) for the classification of drugs and other medical products.

  7. Carbimazole - Wikipedia

    en.wikipedia.org/wiki/Carbimazole

    The onset of anti-thyroid effect is rapid but the onset of clinical effects on thyroid hormone levels in the blood is much slower. This is because the large store of pre-formed T 3 and T 4 in the thyroid gland and bound to thyroid binding globulin (99% bound) has to be depleted before any beneficial clinical effect occurs.