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Takotsubo Cardiomyopathy, Familial Cardiomyopathy, Pre-existing Cardiomyopathy, Valvular Heart Disease, Congenital Heart Disease Peripartum cardiomyopathy (PPCM) is a form of dilated cardiomyopathy that is defined as a deterioration in cardiac function presenting typically between the last month of pregnancy and up to six months postpartum .
High-output heart failure is a heart condition that occurs when the cardiac output is higher than normal because of increased peripheral demand. There is a circulatory overload which may lead to pulmonary edema secondary to an elevated diastolic pressure in the left ventricle .
Severe anemia leads to hyperdynamic circulation, which means high-output cardiac failure causes the blood to circulate rapidly. The excessive pumping of blood causes the left side of the heart to fail leading to pulmonary edema. The build up of fluid in the lungs increases the pressure in the lungs leading to vasoconstriction.
Untreated profound anemia can cause high-output heart failure, with pallor, enlarged liver and/or spleen, generalized swelling, and respiratory distress. [citation needed] HDN can be the cause of hydrops fetalis, an often-severe form of prenatal heart failure that causes fetal edema. [5]
The heart adapts to the increased cardiac demand that occurs during pregnancy in many ways. Cardiac output (Lit./Min.): 6.26; Stroke Volume (Ml.): 75; Heart Rate (Per min.): 85; Blood Pressure: Unaffected; Cardiac output increases throughout early pregnancy, and peaks in the third trimester, usually to 30-50% above baseline. [6]
High-output heart failure can occur when there is increased cardiac demand that results in increased left ventricular diastolic pressure which can develop into pulmonary congestion (pulmonary edema). [46] Several terms are closely related to heart failure and may be the cause of heart failure, but should not be confused with it.
Peripartum cardiomyopathy is a heart failure caused by a decrease in left ventricular ejection fraction (LVEF) to <45% which occurs towards the end of pregnancy or a few months postpartum. Symptoms include shortness of breath in various positions and/or with exertion, fatigue, pedal edema, and chest tightness.
In effort to compensate the heart will increase cardiac output and heart rate, which accounts for the increased pulse pressure and sinus tachycardia. [1] The condition sometimes accompanies septic shock, preeclampsia, and other physiological and psychiatric conditions. [citation needed]