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Progression and build-up of dental plaque can give rise to tooth decay – the localised destruction of the tissues of the tooth by acid produced from the bacterial degradation of fermentable sugar – and periodontal problems such as gingivitis and periodontitis; [3] hence it is important to disrupt the mass of bacteria and remove it. [4]
And gum recession can leave some tooth surfaces without the enamel that protects them from cavities. Women in this phase of life are also more likely to develop periodontal disease, when plaque ...
Tooth decay is caused by biofilm (dental plaque) lying on the teeth and maturing to become cariogenic (causing decay). Certain bacteria in the biofilm produce acids, primarily lactic acid , in the presence of fermentable carbohydrates such as sucrose , fructose , and glucose .
If the socket that holds the tooth is less dense, Sollecito said, it’s more vulnerable to bone loss. And gum recession can leave some tooth surfaces without the enamel that protects them from cavities. Women in this phase of life are also more likely to develop periodontal disease, when plaque and bacteria collect under gums and around teeth.
Once the plaque acid has been neutralized, the minerals can return from the plaque and saliva to the enamel surface. However, the capacity for remineralization is limited, and if sugars enter the mouth too frequently then a net loss of minerals from enamel produces a cavity, through which bacteria can infect the inner tooth and destroy the ...
Examples of secondary causes are those things that, by definition, cause microbic plaque accumulation, such as restoration overhangs and root proximity. The excess restorative material that exceeds the natural contours of restored teeth, such as these, are termed "overhangs", and serve to trap microbic plaque, potentially leading to localized ...
Nope, it's not eating candy.
Plaque that remains in the oral cavity long enough will eventually calcify and become calculus. [18] Calculus is detrimental to gingival health because it serves as a trap for increased plaque formation and retention; thus, calculus, along with other factors that cause a localized build-up of plaque, is referred to as a secondary aetiology of ...