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Cerebral hyperperfusion syndrome, also known as reperfusion syndrome, is a dysregulated state of cerebral blood flow following the restoration of arterial blood flow to the brain, usually following treatment of carotid artery stenosis. [1]
Reperfusion injury, sometimes called ischemia-reperfusion injury (IRI) or reoxygenation injury, is the tissue damage caused when blood supply returns to tissue (re-+ perfusion) after a period of ischemia or lack of oxygen (anoxia or hypoxia).
This leads to poor oxygen supply or cerebral hypoxia and thus leads to the death of brain tissue or cerebral infarction/ischemic stroke. [2] It is a sub-type of stroke along with subarachnoid hemorrhage and intracerebral hemorrhage. [3] Ischemia leads to alterations in brain metabolism, reduction in metabolic rates, and energy crisis. [4]
Following surgery, a rare early complication is cerebral hyperperfusion syndrome, also known as reperfusion syndrome, which is associated with headache and high blood pressure following surgery. Long term complications include restenosis of the endarterectomy bed, although the clinical significance of this is controversial in asymptomatic patients.
Congenital diseases are medical conditions that are present at birth that may be associated with or inherited through genes. [16] Examples of congenital cerebrovascular diseases include arteriovenous malformations, germinal matrix hemorrhage, and CADASIL (cerebral autosomal-dominant arteriopathy with subcortical infarcts and leukoencephalopathy). [9]
Overperfusion and underperfusion should not be confused with hypoperfusion and hyperperfusion, which relate to the perfusion level relative to a tissue's current need to meet its metabolic needs. For example, hypoperfusion can be caused when an artery or arteriole that supplies blood to a volume of tissue becomes blocked by an embolus , causing ...
Cerebral contusion; Intracerebral hemorrhage; Cerebral hyperperfusion syndrome; Cerebral laceration; Cerebral palsy; Cerebral perfusion pressure; Childhood acquired brain injury; Chronic Effects of Neurotrauma Consortium; Chronic traumatic encephalopathy; Closed-head injury; Cognitive rehabilitation therapy; Complications of traumatic brain ...
Cerebral amyloid angiopathy; Cerebral folate deficiency; Cerebral hyperperfusion syndrome; Cerebral hypoxia; Cerebral vasospasm; Cerebritis; Cerebrospinal fluid leak; Childhood acquired brain injury; Cortical visual impairment