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Mitral regurgitation, also known as mitral insufficiency or mitral incompetence, is the backward flow of blood from the left ventricle, through the mitral valve, and into the left atrium, when the left ventricle contracts, resulting in a systolic murmur radiating to the left armpit. [5] [3]
Mitral valve prolapse is frequently associated with mild mitral regurgitation, [15] where blood aberrantly flows from the left ventricle into the left atrium during systole. In the United States , MVP is the most common cause of severe, non-ischemic mitral regurgitation. [ 4 ]
Mitral regurgitation: the backflow of blood from the left ventricle into the left atrium, owing to insufficiency of the mitral valve; it may be acute or chronic, and is usually due to mitral valve prolapse, rheumatic heart disease, or a complication of cardiac dilatation. See also Mitral regurgitation.
Pulmonary regurgitation (stenosis is possible, but rare) Inferior vena cava size as estimate of central venous pressure; Aortic root size for thoracic ascending aortic aneurysm; Pericardial effusion size; All function dysfunction is graded on a scale (normal, trace, mild, moderate, or severe) based on various criteria.
Mitral regurgitation may be treated medically with vasodilators, diuretics, digoxin, antiarrhythmics, and chronic anticoagulation. [15] [16] Mild to moderate mitral regurgitation should be followed with echocardiography and cardiac stress test every 1–3 years. [15]
Anticoagulants: To prevent embolization.. Beta blockers: To block the effects of certain hormones on the heart to slow the heart rate.. Calcium Channel Blockers: Help slow the heart rate by blocking the number of electrical impulses that pass through the AV node into the lower heart chambers (ventricles).
Mitral annular calcification (MAC) is a multifactorial chronic degenerative process in which calcium with lipid is deposited in the annular fibrosa ring of the heart's mitral valve. MAC was first discovered and described in 1908 by M. Bonninger in the journal Deutsche Medizinische Wochenschrift . [ 1 ]
Common causes include aortic or pulmonary regurgitation and left anterior descending artery stenosis. Mid-diastolic murmurs start after S2 and end before S1. They are due to turbulent flow across the atrioventricular (mitral & tricuspid) valves during the rapid filling phase from mitral or tricuspid stenosis.
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