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Within the human body, biofilms are present on the teeth as dental plaque, where they may cause tooth decay and gum disease. These biofilms can either be in an uncalcified state that can be removed by dental instruments, or a calcified state which is more difficult to remove. Removal techniques can also include antimicrobials. [75]
The bacteria found in the biofilm elicit a host response resulting in localized inflammation of the tissue. [22] This is characterized by the cardinal signs of inflammation including a red, puffy appearance of the gums and bleeding due to brushing or flossing. [23] Gingivitis due to plaque can be reversible by removal of the plaque.
The development of biofilm begins with pellicle formation. Pellicle is an acellular proteinaceous film which covers the teeth. Bacteria colonize on the teeth by adhering to the pellicle-coated surface. Over time, a mature biofilm is formed, creating a cariogenic environment on the tooth surface.
The C2DA inhibit methicillin resistant staphylococcus biofilm, but don't eliminate it. The mechanism of the biofilm inhibition by these molecules is still unknown. C2D is a medium of fatty acid chain that effect on staphylococcus aureus biofilm and dispersion of these biofilm. Pseudomonas aeruginosa is the main source for these molecules. [15]
A dynamic equilibrium exists notably between the bacteria of dental plaque and the host's immune system, enabling the plaque to stay behind in the mouth when other biofilms are washed away. [19] In equilibrium, the bacterial biofilm produced by the fermentation of sugar in the mouth is quickly swept away by the saliva, except for dental plaque.
There has been a great effort to develop efficient methods for biofilm removal or prevention in clinical and food manufacturing processes. On the other hand, current research suggested that biofilms can be useful for constructive purposes such as a template of new materials [1] [2] that may find applications in the industry. One distinctive ...
He also studies biofilms elsewhere in the human body, including prosthetic joint, middle ear, and sinus infections. [1] [5] Jakubovics, Alexander H. Rickard, Derek Samarian, and Ethan Kolderman jointly hold a patent for the use of L-arginine to remove dental plaque. [6]
Moreover, S. epidermidis biofilm formation by releasing the exopolysaccharide intercellular adhesion (PIA) provides the susceptible anaerobic environment to P. acnes colonisation and protects it from the innate human immunity molecules. [23] Both P. acnes and S. epidermidis can interact to protect the host skin health from pathogens ...