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The biological model of abnormality (the only model not based on psychological principles) is based on the assumptions that if the brain, neuroanatomy and related biochemicals are all physical entities and work together to mediate psychological processes, then treating any mental abnormality must be physical/biological.
Instead of viewing schizophrenia as a by-product of brain evolution, one model presents schizophrenia to be one extreme of a sexually selected fitness indicator. [26] This model hypothesizes schizophrenia to be a side effect in sexual selection for certain traits. Sexual selection concerns the mating choices of humans and other animals.
The dopamine hypothesis of schizophrenia or the dopamine hypothesis of psychosis is a model that attributes the positive symptoms of schizophrenia to a disturbed and hyperactive dopaminergic signal transduction. The model draws evidence from the observation that a large number of antipsychotics have dopamine-receptor antagonistic effects. The ...
The common dopamine and glutamate models proposed are not mutually exclusive; each is seen to have a role in the neurobiology of schizophrenia. [128] The most common model put forward was the dopamine hypothesis of schizophrenia, which attributes psychosis to the mind's faulty interpretation of the misfiring of dopaminergic neurons. [129]
Cognitive symptoms of schizophrenia involve disturbances in executive functions, working memory impairment, and inability to sustain attention. [1] Given the high numbers of individuals diagnosed with schizophrenia (nearly 1% of modern-day populations), it is unlikely that the disorder has arisen solely from random mutations. [2]
The causes of schizophrenia that underlie the development of schizophrenia, a psychiatric disorder, are complex and not clearly understood.A number of hypotheses including the dopamine hypothesis, and the glutamate hypothesis have been put forward in an attempt to explain the link between altered brain function and the symptoms and development of schizophrenia.
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