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Those who use lithium should receive regular serum level tests and should monitor thyroid and kidney function for abnormalities, as it interferes with the regulation of sodium and water levels in the body, and can cause dehydration. Dehydration, which is compounded by heat, can result in increasing lithium levels.
Moderate symptoms include confusion, an increased heart rate, and low muscle tone occur at a level of 2.5 to 3.5 mEq/L. [1] Severe symptoms include coma, seizures, low blood pressure and increased body temperature which occur at a lithium concentration greater than 3.5 mEq/L. [1] When lithium overdoses produce neurological deficits or cardiac ...
The extraction of lithium carbonate at high pressures of CO 2 and its precipitation upon depressurizing is the basis of the Quebec process. Lithium carbonate can also be purified by exploiting its diminished solubility in hot water. Thus, heating a saturated aqueous solution causes crystallization of Li 2 CO 3. [20]
More adverse effects including chaotic cardiac rhythm and brain-wave activity with seizures may also occur when lithium concentration in serum increases to 2 mmol/L. Prolonged use of lithium may damage the body's ability to respond properly to hormone vasopressin (ADH), which stimulates water reabsorption.
Generally, seizures are observed in patients who do not have epilepsy. [1] There are many causes of seizures. Organ failure, medication and medication withdrawal, cancer, imbalance of electrolytes, hypertensive encephalopathy, may be some of its potential causes. [2]
The most obvious cause is a kidney or systemic disorder, including amyloidosis, [2] polycystic kidney disease, [3] electrolyte imbalance, [4] [5] or some other kidney defect. [2] The major causes of acquired nephrogenic diabetes insipidus that produce clinical symptoms (e.g., polyuria) in the adult are lithium toxicity and high blood calcium.