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These systemic effects are caused by a traumatic rhabdomyolysis. As muscle cells die, they absorb sodium, water, and calcium; the rhabdomyolysis releases potassium, myoglobin, phosphate, thromboplastin, creatine, and creatine kinase. [citation needed] Crush syndrome can directly come from compartment syndrome, if the injury is left untreated. [8]
For moderate to severe rhabdo cases, patients may need IV fluids and hospital admission. IV fluids help flush out the muscle proteins and electrolytes, and can prevent dangerous heart rhythms and ...
Hospitalization and IV hydration should be the first step in any patient suspected of having myoglobinuria or rhabdomyolysis. The goal is to induce a brisk diuresis to prevent myoglobin precipitation and deposition, which can cause acute kidney injury. Mannitol can be added to assist with diuresis.
Rhabdomyolysis (shortened as rhabdo) is a condition in which damaged skeletal muscle breaks down rapidly, often due to high intensity exercise over a short period. [6] [4] [5] Symptoms may include muscle pains, weakness, vomiting, and confusion. [3] [4] There may be tea-colored urine or an irregular heartbeat.
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Exertional rhabdomyolysis, the exercise-induced muscle breakdown that results in muscle pain/soreness, is commonly diagnosed using the urine myoglobin test accompanied by high levels of creatine kinase (CK).
Treatment may include physical therapy or, if that fails, surgery. [1] ACS is an emergency, and outcome largely depends on the time to diagnosis and treatment. [12] If treated within 3 hours, the prognosis is favorable. [12] Complications and permanent damage can occur. [13] ACS occurs in about 1-10% of those with a tibial shaft fracture. [6]
[citation needed] Comorbidity of cardiomyopathy, arrhythmias [3] and rhabdomyolysis are extremely common in patients under 1 year old which can lead to complications later in life [citation needed]. Loss of awareness or seizure can occur from hypoketotic hypoglycemia, [ 3 ] which is often fatal if not caught in screening.